相似文献
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Molecular characterization of a common fragile site (FRA7H) on human chromosome 7 by the cloning of a simian virus 40 integration site.通过克隆猿猴病毒40整合位点对人类7号染色体上一个常见脆性位点（FRA7H）进行分子特征分析。
Proc Natl Acad Sci U S A. 1998 Jul 7;95(14):8141-6. doi: 10.1073/pnas.95.14.8141.
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Replication delay along FRA7H, a common fragile site on human chromosome 7, leads to chromosomal instability.沿着人类7号染色体上的一个常见脆性位点FRA7H的复制延迟会导致染色体不稳定。
Mol Cell Biol. 2000 Jun;20(12):4420-7. doi: 10.1128/MCB.20.12.4420-4427.2000.
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Direct cloning of DNA sequences from the common fragile site region at chromosome band 3p14.2.直接克隆来自染色体3p14.2带区常见脆性位点区域的DNA序列。
Genomics. 1996 Jul 1;35(1):109-17. doi: 10.1006/geno.1996.0329.
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Human chromosome fragility.人类染色体脆性
Biochim Biophys Acta. 2008 Jan;1779(1):3-16. doi: 10.1016/j.bbagrm.2007.10.005. Epub 2007 Dec 3.
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FRA7G extends over a broad region: coincidence of human endogenous retroviral sequences (HERV-H) and small polydispersed circular DNAs (spcDNA) and fragile sites.FRA7G延伸至一个广泛区域：人类内源性逆转录病毒序列（HERV-H）、小分散环状DNA（spcDNA）与脆性位点的重合。
Oncogene. 1998 May 7;16(18):2311-9. doi: 10.1038/sj.onc.1200202.
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Preferential integration of marker DNA into the chromosomal fragile site at 3p14: an approach to cloning fragile sites.标记DNA优先整合到3p14染色体脆性位点：一种克隆脆性位点的方法。
Proc Natl Acad Sci U S A. 1991 Aug 1;88(15):6657-61. doi: 10.1073/pnas.88.15.6657.
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Direct cloning and analysis of DNA sequences from a region of the Chinese hamster genome associated with aphidicolin-sensitive fragility.直接克隆和分析来自中国仓鼠基因组中与阿非迪霉素敏感性脆性相关区域的DNA序列。
J Cell Sci. 1998 Jun;111 ( Pt 12):1623-34. doi: 10.1242/jcs.111.12.1623.
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FRA3B extends over a broad region and contains a spontaneous HPV16 integration site: direct evidence for the coincidence of viral integration sites and fragile sites.FRA3B延伸至一个广泛区域，并包含一个自发的人乳头瘤病毒16型整合位点：病毒整合位点与脆性位点重合的直接证据。
Hum Mol Genet. 1996 Feb;5(2):187-95. doi: 10.1093/hmg/5.2.187.
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Cloning and characterization of the common fragile site FRA6F harboring a replicative senescence gene and frequently deleted in human tumors.包含一个复制性衰老基因且在人类肿瘤中经常缺失的常见脆性位点FRA6F的克隆与特性分析。
Oncogene. 2002 Oct 17;21(47):7266-76. doi: 10.1038/sj.onc.1205573.
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Aphidicolin-induced FRA3B breakpoints cluster in two distinct regions.阿非科林诱导的FRA3B断点聚集在两个不同区域。
Genomics. 1997 May 1;41(3):485-8. doi: 10.1006/geno.1997.4690.
引用本文的文献
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A predictive chromatin architecture nexus regulates transcription and DNA damage repair.一种预测性染色质结构关联体调控转录和DNA损伤修复。
J Biol Chem. 2025 Mar;301(3):108300. doi: 10.1016/j.jbc.2025.108300. Epub 2025 Feb 11.
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Replication of [AT/TA] Microsatellite Sequences by Human DNA Polymerase δ Holoenzymes Is Dependent on dNTP and RPA Levels.人类 DNA 聚合酶 δ 全酶通过 [AT/TA] 微卫星序列的复制依赖于 dNTP 和 RPA 水平。
Biochemistry. 2024 Apr 16;63(8):969-983. doi: 10.1021/acs.biochem.4c00006. Epub 2024 Mar 26.
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Replication of [AT/TA] microsatellite sequences by human DNA polymerase δ holoenzymes is dependent on dNTP and RPA levels.人DNA聚合酶δ全酶对[AT/TA]微卫星序列的复制依赖于脱氧核苷三磷酸（dNTP）和复制蛋白A（RPA）的水平。
bioRxiv. 2023 Nov 8:2023.11.07.566133. doi: 10.1101/2023.11.07.566133.
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Insights into common fragile site instability: DNA replication challenges at DNA repeat sequences.常见脆弱部位不稳定性的研究进展：DNA 重复序列处的 DNA 复制挑战。
Emerg Top Life Sci. 2023 Dec 14;7(3):277-287. doi: 10.1042/ETLS20230023.
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Mitotic DNA synthesis in response to replication stress requires the sequential action of DNA polymerases zeta and delta in human cells.有丝分裂 DNA 合成对复制应激的反应需要 DNA 聚合酶 zeta 和 delta 在人类细胞中的顺序作用。
Nat Commun. 2023 Feb 9;14(1):706. doi: 10.1038/s41467-023-35992-5.
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Fragile sites, chromosomal lesions, tandem repeats, and disease.脆性位点、染色体病变、串联重复序列与疾病。
Front Genet. 2022 Nov 17;13:985975. doi: 10.3389/fgene.2022.985975. eCollection 2022.
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First Comprehensive Characterization of Phayre's Leaf-Monkey () Karyotype.菲氏叶猴核型的首次全面表征
Front Genet. 2022 Mar 11;13:841681. doi: 10.3389/fgene.2022.841681. eCollection 2022.
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Alternative DNA Structures : Molecular Evidence and Remaining Questions.备选 DNA 结构：分子证据与未解问题
Microbiol Mol Biol Rev. 2020 Dec 23;85(1). doi: 10.1128/MMBR.00110-20. Print 2021 Feb 17.
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Regulation of breast cancer metastasis signaling by miRNAs.miRNAs 调控乳腺癌转移信号通路
Cancer Metastasis Rev. 2020 Sep;39(3):837-886. doi: 10.1007/s10555-020-09905-7.
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Impaired Replication Timing Promotes Tissue-Specific Expression of Common Fragile Sites.复制定时障碍促进常见脆弱部位的组织特异性表达。
Genes (Basel). 2020 Mar 19;11(3):326. doi: 10.3390/genes11030326.
本文引用的文献
1
Replication of a common fragile site, FRA3B, occurs late in S phase and is delayed further upon induction: implications for the mechanism of fragile site induction.常见脆性位点FRA3B的复制发生在S期晚期，诱导后会进一步延迟：对脆性位点诱导机制的启示。
Hum Mol Genet. 1998 Apr;7(4):755-61. doi: 10.1093/hmg/7.4.755.
2
Fish mapping of YAC clones at human chromosomal band 7q31.2: identification of YACS spanning FRA7G within the common region of LOH in breast and prostate cancer.人类染色体7q31.2处YAC克隆的荧光原位杂交定位：在乳腺癌和前列腺癌中杂合性缺失的共同区域内跨越FRA7G的YAC克隆的鉴定
Genes Chromosomes Cancer. 1998 Feb;21(2):152-9. doi: 10.1002/(sici)1098-2264(199802)21:2<152::aid-gcc11>3.0.co;2-t.
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Sequence of the FRA3B common fragile region: implications for the mechanism of FHIT deletion.FRA3B常见脆性区域的序列：对FHIT基因缺失机制的启示
Proc Natl Acad Sci U S A. 1997 Dec 23;94(26):14584-9. doi: 10.1073/pnas.94.26.14584.
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Trinucleotide repeats affect DNA replication in vivo.三核苷酸重复序列在体内影响DNA复制。
Nat Genet. 1997 Nov;17(3):298-304. doi: 10.1038/ng1197-298.
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Aphidicolin-induced FRA3B breakpoints cluster in two distinct regions.阿非科林诱导的FRA3B断点聚集在两个不同区域。
Genomics. 1997 May 1;41(3):485-8. doi: 10.1006/geno.1997.4690.
6
DNA secondary structures and the evolution of hypervariable tandem arrays.DNA二级结构与高变串联阵列的进化
J Biol Chem. 1997 Apr 4;272(14):9517-23. doi: 10.1074/jbc.272.14.9517.
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Positions of chromosome 3p14.2 fragile sites (FRA3B) within the FHIT gene.FHIT基因内3号染色体3p14.2脆性位点（FRA3B）的位置。
Cancer Res. 1997 Mar 15;57(6):1166-70.
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Chromosome 3p14 homozygous deletions and sequence analysis of FRA3B.3号染色体p14区域纯合缺失及脆性位点FRA3B的序列分析
Hum Mol Genet. 1997 Feb;6(2):193-203. doi: 10.1093/hmg/6.2.193.
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Human chromosomal fragile site FRA16B is an amplified AT-rich minisatellite repeat.人类染色体脆性位点FRA16B是一个扩增的富含AT的小卫星重复序列。
Cell. 1997 Feb 7;88(3):367-74. doi: 10.1016/s0092-8674(00)81875-9.
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Long CCG triplet repeat blocks exclude nucleosomes: a possible mechanism for the nature of fragile sites in chromosomes.长CCG三联体重复序列块排除核小体：染色体中脆性位点性质的一种可能机制。
J Mol Biol. 1996 Nov 8;263(4):511-6. doi: 10.1006/jmbi.1996.0593.
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