Glaros A G, Tabacchi K N, Glass E G
Department of Dental Public Health and Behavioral Science, School of Dentistry, University of Missouri-Kansas City, USA.
J Orofac Pain. 1998 Spring;12(2):145-52.
Parafunctional activities are assumed to play an important role in temporomandibular disorders (TMD), but experimental data in support of this hypothesis are lacking. This study examined the role of parafunctional clenching on various measures of TMD pain. Five subjects participated in daily 17-minute electromyogram biofeedback training sessions structured in three phases. Subjects were instructed to maintain temporalis and masseter muscle activity below 2 microV in the first (decrease) phase of training (10 sessions), above 10 microV in the second (increase) phase (1 to 8 sessions), and below 2 microV in the third (decrease) phase (10 to 15 sessions). Preliminary screening examinations showed that none of the subjects had TMD. Two subjects reported intolerable pain during increase training, and both were diagnosed with a TMD during this phase. No subject was diagnosed with TMD pain during either decrease training phase. The authors conclude that chronic, low-level parafunctional clenching may be a factor in the cause of TMD pain.
人们认为副功能活动在颞下颌关节紊乱病（TMD）中起重要作用，但缺乏支持这一假设的实验数据。本研究探讨了副功能紧咬牙对TMD疼痛各项指标的影响。五名受试者参加了为期17分钟的每日肌电图生物反馈训练课程，该课程分为三个阶段。在训练的第一阶段（降低阶段，共10节课程），受试者被要求将颞肌和咬肌活动维持在2微伏以下；在第二阶段（增加阶段，共1至8节课程），活动维持在10微伏以上；在第三阶段（降低阶段，共10至15节课程），活动维持在2微伏以下。初步筛查检查显示，所有受试者均无TMD。两名受试者在增加训练期间报告了无法忍受的疼痛，两人在此阶段均被诊断为患有TMD。在两个降低训练阶段，均无受试者被诊断为TMD疼痛。作者得出结论，慢性、低水平副功能紧咬牙可能是TMD疼痛的一个致病因素。
