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Ceramide transport from endoplasmic reticulum to Golgi apparatus is not vesicle-mediated.神经酰胺从内质网到高尔基体的转运不是由囊泡介导的。
Biochem J. 1998 Aug 1;333 ( Pt 3)(Pt 3):779-86. doi: 10.1042/bj3330779.
2
Genetic evidence for ATP-dependent endoplasmic reticulum-to-Golgi apparatus trafficking of ceramide for sphingomyelin synthesis in Chinese hamster ovary cells.中国仓鼠卵巢细胞中用于鞘磷脂合成的神经酰胺经ATP依赖的内质网到高尔基体转运的遗传学证据。
J Cell Biol. 1999 Feb 22;144(4):673-85. doi: 10.1083/jcb.144.4.673.
3
Vesicular and nonvesicular transport of ceramide from ER to the Golgi apparatus in yeast.酵母中神经酰胺从内质网到高尔基体的囊泡运输和非囊泡运输
J Cell Biol. 2001 Dec 10;155(6):949-59. doi: 10.1083/jcb.200105033. Epub 2001 Dec 3.
4
Ceramide excluded from cell-free vesicular lipid transfer from endoplasmic reticulum to Golgi apparatus. Evidence for lipid sorting.神经酰胺被排除在从内质网到高尔基体的无细胞囊泡脂质转运之外。脂质分选的证据。
Biochim Biophys Acta. 1993 Feb 23;1146(1):9-16. doi: 10.1016/0005-2736(93)90332-t.
5
Reconstitution of ATP- and cytosol-dependent transport of de novo synthesized ceramide to the site of sphingomyelin synthesis in semi-intact cells.在半完整细胞中，将新合成的神经酰胺进行ATP和胞质溶胶依赖性转运至鞘磷脂合成位点的重建。
J Biol Chem. 2000 Sep 29;275(39):29938-45. doi: 10.1074/jbc.M004470200.
6
A novel inhibitor of ceramide trafficking from the endoplasmic reticulum to the site of sphingomyelin synthesis.一种从内质网到鞘磷脂合成部位的神经酰胺转运新型抑制剂。
J Biol Chem. 2001 Nov 23;276(47):43994-4002. doi: 10.1074/jbc.M104884200. Epub 2001 Sep 6.
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Molecular mechanisms and regulation of ceramide transport.神经酰胺转运的分子机制与调控
Biochim Biophys Acta. 2005 Jun 1;1734(3):220-34. doi: 10.1016/j.bbalip.2005.04.001.
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Sphingosine-1-phosphate phosphohydrolase regulates endoplasmic reticulum-to-golgi trafficking of ceramide.鞘氨醇-1-磷酸磷酸水解酶调节神经酰胺从内质网到高尔基体的运输。
Mol Cell Biol. 2006 Jul;26(13):5055-69. doi: 10.1128/MCB.02107-05.
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Limonoid compounds inhibit sphingomyelin biosynthesis by preventing CERT protein-dependent extraction of ceramides from the endoplasmic reticulum.柠檬苦素类化合物通过阻止 CERT 蛋白依赖性从内质网提取神经酰胺来抑制鞘磷脂生物合成。
J Biol Chem. 2012 Jul 13;287(29):24397-411. doi: 10.1074/jbc.M112.344432. Epub 2012 May 17.
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Cell-free transfer of the vesicular stomatitis virus G protein from an endoplasmic reticulum compartment of baby hamster kidney cells to a rat liver Golgi apparatus compartment for Man8-9 to Man5 processing.水泡性口炎病毒G蛋白从幼仓鼠肾细胞的内质网区室向大鼠肝脏高尔基体区室的无细胞转移，用于Man8 - 9到Man5的加工。
Arch Biochem Biophys. 1999 Jul 15;367(2):265-73. doi: 10.1006/abbi.1999.1276.
引用本文的文献
1
Role of Intracellular Lipid Logistics in the Preferential Usage of Very Long Chain-Ceramides in Glucosylceramide.细胞内脂质物流在葡萄糖神经酰胺中极长链神经酰胺优先利用中的作用
Int J Mol Sci. 2016 Oct 21;17(10):1761. doi: 10.3390/ijms17101761.
2
Ceramide transfer protein and cancer.神经酰胺转移蛋白与癌症。
Anticancer Agents Med Chem. 2011 Nov;11(9):904-10. doi: 10.2174/187152011797655087.
3
The ceramide-enriched trans-Golgi compartments reorganize together with other parts of the Golgi apparatus in response to ATP-depletion.富含神经酰胺的反式高尔基体隔室与高尔基体其他部分一起在 ATP 耗竭时发生重排。
Histochem Cell Biol. 2011 Feb;135(2):159-71. doi: 10.1007/s00418-010-0773-z. Epub 2011 Jan 12.
4
Lipid trafficking sans vesicles: where, why, how?无囊泡脂质运输：何处、为何、如何？
Cell. 2010 Dec 10;143(6):870-4. doi: 10.1016/j.cell.2010.11.031.
5
Acute perturbations in Golgi organization impact de novo sphingomyelin synthesis.高尔基体组织的急性扰动会影响鞘磷脂的从头合成。
Traffic. 2008 Nov;9(11):1894-904. doi: 10.1111/j.1600-0854.2008.00810.x. Epub 2008 Aug 9.
6
Ceramide trafficking: learning from the rebels.
J Biosci. 2004 Sep;29(3):209-10. doi: 10.1007/BF02702600.
7
Glycosphingolipids and cell death.糖鞘脂与细胞死亡。
Glycoconj J. 2004;20(1):39-47. doi: 10.1023/B:GLYC.0000016741.88476.8b.
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The mitochondria-associated endoplasmic-reticulum subcompartment (MAM fraction) of rat liver contains highly active sphingolipid-specific glycosyltransferases.大鼠肝脏的线粒体相关内质网亚区室（MAM组分）含有高活性的鞘脂特异性糖基转移酶。
Biochem J. 2003 May 1;371(Pt 3):1013-9. doi: 10.1042/BJ20021834.
9
Identification of ceramide binding proteins in neuronal cells: a critical point of view.神经元细胞中神经酰胺结合蛋白的鉴定：批判性观点
Neurochem Res. 2002 Aug;27(7-8):717-27. doi: 10.1023/a:1020288403626.
10
Sphingosine-1-phosphate phosphohydrolase in regulation of sphingolipid metabolism and apoptosis.鞘氨醇-1-磷酸磷酸水解酶在鞘脂代谢和细胞凋亡调控中的作用
J Cell Biol. 2002 Sep 16;158(6):1039-49. doi: 10.1083/jcb.200203123.
本文引用的文献
1
A rapid method of total lipid extraction and purification.一种快速的总脂质提取与纯化方法。
Can J Biochem Physiol. 1959 Aug;37(8):911-7. doi: 10.1139/o59-099.
2
Role of NAD+ and ADP-ribosylation in the maintenance of the Golgi structure.NAD⁺与ADP核糖基化在高尔基体结构维持中的作用。
J Cell Biol. 1997 Dec 1;139(5):1109-18. doi: 10.1083/jcb.139.5.1109.
3
Characterization of ceramide synthesis. A dihydroceramide desaturase introduces the 4,5-trans-double bond of sphingosine at the level of dihydroceramide.神经酰胺合成的特征。二氢神经酰胺去饱和酶在二氢神经酰胺水平引入鞘氨醇的4,5-反式双键。
J Biol Chem. 1997 Sep 5;272(36):22432-7. doi: 10.1074/jbc.272.36.22432.
4
Dihydroceramide biology. Structure-specific metabolism and intracellular localization.二氢神经酰胺生物学。结构特异性代谢与细胞内定位。
J Biol Chem. 1997 Aug 22;272(34):21128-36. doi: 10.1074/jbc.272.34.21128.
5
Transport of sphingomyelin to the cell surface is inhibited by brefeldin A and in mitosis, where C6-NBD-sphingomyelin is translocated across the plasma membrane by a multidrug transporter activity.鞘磷脂向细胞表面的转运受到布雷菲德菌素A的抑制，并且在有丝分裂过程中，C6-NBD-鞘磷脂通过一种多药转运蛋白活性跨质膜转运。
J Cell Sci. 1997 Jan;110 ( Pt 1):75-83. doi: 10.1242/jcs.110.1.75.
6
Protein trafficking along the exocytotic pathway.蛋白质沿胞吐途径的运输。
Bioessays. 1993 Apr;15(4):231-8. doi: 10.1002/bies.950150403.
7
The long-chain sphingoid base of sphingolipids is acylated at the cytosolic surface of the endoplasmic reticulum in rat liver.在大鼠肝脏中，鞘脂的长链鞘氨醇碱在内质网的胞质表面被酰化。
Biochem J. 1993 Mar 15;290 ( Pt 3)(Pt 3):751-7. doi: 10.1042/bj2900751.
8
Ceramide excluded from cell-free vesicular lipid transfer from endoplasmic reticulum to Golgi apparatus. Evidence for lipid sorting.神经酰胺被排除在从内质网到高尔基体的无细胞囊泡脂质转运之外。脂质分选的证据。
Biochim Biophys Acta. 1993 Feb 23;1146(1):9-16. doi: 10.1016/0005-2736(93)90332-t.
9
Differential metabolism and trafficking of sphingolipids in differentiated versus undifferentiated HT29 cells.分化与未分化的HT29细胞中鞘脂的差异代谢与转运
Int J Cancer. 1993 Jul 9;54(5):839-45. doi: 10.1002/ijc.2910540519.
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