Krystkowiak P, Vermersch P, Beaucaire G, Petit H
Clinique Neurologique, CHRU de Lille.
Rev Neurol (Paris). 1997 Dec;153(12):781-4.
A 59 year-old-man had ataxia and hemiparesis of progressive onset which resolved in a few weeks. CT scan and magnetic resonance imaging showed white matter multiple lesions. The cerebrospinal fluid was consistent with an inflammatory disease. The diagnosis of multiple sclerosis was evoked. However, the patient had chronic brucellosis and serological rates increased and decreased according to the clinical and radiological signs. Thus, encephalitis caused by brucellosis was evoked. The mechanism is assumed to be a demyelinating process caused by a deep focus reactivation.
一名59岁男性出现进行性共济失调和偏瘫，数周后症状缓解。CT扫描和磁共振成像显示白质有多处病变。脑脊液检查结果符合炎症性疾病表现。曾考虑多发性硬化症的诊断。然而，该患者患有慢性布鲁氏菌病，血清学指标随临床和影像学表现而升高和降低。因此，考虑为布鲁氏菌病所致脑炎。其机制推测为深部病灶重新激活引起的脱髓鞘过程。
