Roeva L A, Chubarova E Ia
Med Tekh. 1998 May-Jun(3):30-3.
The paper analyzes left ventricular structure-fraction relationships in the development of postinfarct aneurysm. The altered internal architectonics induces to systemic hemodynamic changes, drastically elevated intraventricular pressure. This is caused by to the dysfunction of the papillary-trabecular complex in the left ventricular cavity, which in turn. This leads to the fact that the heart work as a positive-displacement pump, by losing its capacity as a centrifugal component, by making the myocardium require additional energy expenditures, which in turn appears as varying heart failure. This investigation is of definite practical value in developing adequate correction methods for postinfarct aneurysm which can occur with the retained or formed certain ratios of cardiac structures.
本文分析了心肌梗死后室壁瘤形成过程中左心室结构与分数之间的关系。内部结构的改变会引发全身血流动力学变化，导致心室内压力急剧升高。这是由左心室腔内乳头肌 - 小梁复合体功能障碍引起的，进而导致心脏作为容积泵工作，失去其作为离心组件的功能，使心肌需要额外的能量消耗，进而表现为不同程度的心力衰竭。这项研究对于制定针对心肌梗死后室壁瘤的适当矫正方法具有一定的实际价值，心肌梗死后室壁瘤可能在心脏结构保持或形成特定比例时出现。
