Gupta S, Prabhu M R, Gupta M S, Niblett D
Department of Anaesthesia, Bedford Hospital, UK.
Eur J Anaesthesiol. 1998 Sep;15(5):603-6. doi: 10.1046/j.1365-2346.1998.00364.x.
We report a case of non-ketotic hyperosmolar coma, which presented with blood sugar levels far exceeding any previously recorded in the literature. The patient developed acute renal failure, probably because of rhabdomyolysis, which was successfully managed with continuous veno-venous haemofiltration. He developed blurring of vision resulting from biochemical changes, which was managed conservatively. We discuss the mechanisms of causation of the renal failure and visual blurring.
我们报告一例非酮症高渗性昏迷病例,其血糖水平远超文献中既往记录的任何数值。患者出现急性肾衰竭,可能是由于横纹肌溶解所致,通过持续静脉-静脉血液滤过成功处理。他因生化改变出现视力模糊,采取保守治疗。我们讨论了肾衰竭和视力模糊的发病机制。