小鼠联合治疗：我们能学到什么有助于理解类风湿性关节炎的知识？ - Suppr

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超能文献

Combination therapy in mice: what can we learn that may be useful for understanding rheumatoid arthritis?

作者信息

Williams R O

机构信息

Kennedy Institute of Rheumatology, London, UK.

出版信息

Springer Semin Immunopathol. 1998;20(1-2):165-80. doi: 10.1007/BF00832005.

DOI:10.1007/BF00832005

PMID:9836375

Abstract

摘要

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Combination therapy in mice: what can we learn that may be useful for understanding rheumatoid arthritis?小鼠联合治疗：我们能学到什么有助于理解类风湿性关节炎的知识？

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Anti-tumour necrosis factor specific antibody (infliximab) treatment provides insights into the pathophysiology of rheumatoid arthritis.抗肿瘤坏死因子特异性抗体（英夫利昔单抗）治疗为类风湿关节炎的病理生理学提供了深入见解。

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Anti-TNF alpha therapy in rheumatoid arthritis--current and future directions.类风湿关节炎中的抗肿瘤坏死因子α治疗——现状与未来方向

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Nimesulide improves the symptomatic and disease modifying effects of leflunomide in collagen induced arthritis.尼美舒利可改善来氟米特对胶原诱导性关节炎的症状缓解和病情改善作用。

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Regulatory aspects of evaluating combination treatments in autoimmune diseases.

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Tumour necrosis factor-alpha (TNF-alpha) enhances lymphocyte migration into rheumatoid synovial tissue transplanted into severe combined immunodeficient (SCID) mice.肿瘤坏死因子-α（TNF-α）增强淋巴细胞向移植到严重联合免疫缺陷（SCID）小鼠体内的类风湿性滑膜组织的迁移。

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Therapeutic actions of cyclosporine and anti-tumor necrosis factor alpha in collagen-induced arthritis and the effect of combination therapy.环孢素和抗肿瘤坏死因子α在胶原诱导性关节炎中的治疗作用及联合治疗的效果。

Arthritis Rheum. 1998 Oct;41(10):1806-12. doi: 10.1002/1529-0131(199810)41:10<1806::AID-ART12>3.0.CO;2-9.

J Immunol. 1997 Dec 15;159(12):6253-9.

Suppression of collagen-induced arthritis by continuous administration of IL-4.通过持续给予白细胞介素-4抑制胶原诱导的关节炎。

J Immunol. 1997 Dec 1;159(11):5687-96.

Blockade of IL-12 during the induction of collagen-induced arthritis (CIA) markedly attenuates the severity of the arthritis.在胶原诱导的关节炎（CIA）诱导过程中阻断白细胞介素-12可显著减轻关节炎的严重程度。

Clin Exp Immunol. 1998 Feb;111(2):377-83. doi: 10.1046/j.1365-2249.1998.00485.x.

CD40 and its ligand in cell-mediated immunity.细胞介导免疫中的CD40及其配体。

Agents Actions Suppl. 1998;49:17-22. doi: 10.1007/978-3-0348-8857-8_4.

Treatment of a newly established transgenic model of chronic arthritis with nondepleting anti-CD4 monoclonal antibody.用非耗竭性抗CD4单克隆抗体治疗新建立的慢性关节炎转基因模型。

J Immunol. 1997 Nov 15;159(10):5032-41.

Long-term inhibition of murine lupus by brief simultaneous blockade of the B7/CD28 and CD40/gp39 costimulation pathways.通过短暂同时阻断B7/CD28和CD40/gp39共刺激途径对小鼠狼疮进行长期抑制。

J Immunol. 1997 Oct 1;159(7):3104-8.

The Dunlop-Dottridge Lecture: prognosis in inflammatory arthritis: the value of HLA genotyping and the oncological analogy.

J Rheumatol. 1997 Jul;24(7):1436-42.

Conversion in vivo from an early dominant Th0/Th1 response to a Th2 phenotype during the development of collagen-induced arthritis.在胶原诱导性关节炎发展过程中，体内早期占主导的Th0/Th1反应向Th2表型的转变。

Eur J Immunol. 1997 Jun;27(6):1451-8. doi: 10.1002/eji.1830270623.

Treatment with depleting CD4 monoclonal antibody results in a preferential loss of circulating naive T cells but does not affect IFN-gamma secreting TH1 cells in humans.用耗竭性CD4单克隆抗体进行治疗会导致循环中的初始T细胞优先丢失，但不影响人类分泌干扰素-γ的TH1细胞。

J Clin Invest. 1997 May 1;99(9):2225-31. doi: 10.1172/JCI119396.

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