Maestracci P, Grimaud D
Ann Anesthesiol Fr. 1975;16 Spec No 2-3:101-10.
Patients who are victims of near drowning in fresh water or salt water very frequently have acute edema of the lung which occurs either immediately, or after a free interval of variable duration. The mechanism of this edema is explained, in near drowning in salt water, by the hyperosmolarity of the alveolar fluid leading to a seeping of plasma from the capillaries in the alveoli. In the case of near drowning in fresh water, it is on the contrary the inhaled liquid which passes into the circulation, therby leading to immediate hypervolemia, but this overload is only transitory and is not responsible for the pulmonary edema which occurs later is not accompanied by a rise in pulmonary capillary pressure. It is therefore a lesional edema as is certified by the anatomopathological modifications found in the lungs of drowned patients. Therapeutic management must therefore take into consideration this physiopathology of acute edema of the lung in the drowned.
淡水或海水溺水的患者经常会出现急性肺水肿,这种肺水肿要么立即发生,要么在一段时长不定的无明显症状期后出现。在海水溺水的情况下,这种肺水肿的机制是肺泡液的高渗性导致血浆从肺泡中的毛细血管渗出。相反,在淡水溺水的情况下,是吸入的液体进入循环,从而导致立即出现血容量过多,但这种过载只是暂时的,并非导致随后发生的肺水肿的原因,因为随后发生的肺水肿并未伴有肺毛细血管压力升高。因此,这是一种病变性水肿,溺水患者肺部的解剖病理学改变证实了这一点。因此,治疗管理必须考虑到溺水患者急性肺水肿的这种生理病理学情况。