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Mechanisms of ethanol-induced impairment in asialoglycoprotein receptor expression and function.

作者信息

Ishii H

出版信息

J Gastroenterol. 1998 Dec;33(6):920-1. doi: 10.1007/s005350050203.

DOI:10.1007/s005350050203
PMID:9853575
Abstract
摘要

相似文献

1
Mechanisms of ethanol-induced impairment in asialoglycoprotein receptor expression and function.乙醇诱导去唾液酸糖蛋白受体表达和功能损伤的机制。
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Suppressive effect of ethanol on the expression of hepatic asialoglycoprotein receptors augmented by interleukin-1beta, interleukin-6, and tumor necrosis factor-alpha.
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Ethanol inhibits asialoglycoprotein receptor synthesis but augments its mRNA expression in a human hepatoma cell line.乙醇抑制去唾液酸糖蛋白受体的合成,但增强其在人肝癌细胞系中的mRNA表达。
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Regulation of asialoglycoprotein receptor synthesis by inflammation-related cytokines in HepG2 cells.
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[Analysis of experiments on kinetics of recycling of asialoglycoprotein receptors on the basis of a mathematical model].基于数学模型的去唾液酸糖蛋白受体循环动力学实验分析
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Effects of ethanol on receptor-mediated endocytosis in the liver.乙醇对肝脏中受体介导的内吞作用的影响。
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Decreased binding of asialoglycoproteins to hepatocytes from ethanol-fed rats. Consequence of both impaired synthesis and inactivation of the asialoglycoprotein receptor.
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引用本文的文献

1
Constant serum levels of secreted asialoglycoprotein receptor sH2a and decrease with cirrhosis.血清中分泌型去唾液酸糖蛋白受体 sH2a 的水平持续下降,与肝硬化有关。
World J Gastroenterol. 2011 Dec 28;17(48):5305-9. doi: 10.3748/wjg.v17.i48.5305.
2
A secreted form of the asialoglycoprotein receptor, sH2a, as a novel potential noninvasive marker for liver fibrosis.一种分泌型的去唾液酸糖蛋白受体(sH2a),作为一种新型潜在的非侵入性肝纤维化标志物。
PLoS One. 2011;6(11):e27210. doi: 10.1371/journal.pone.0027210. Epub 2011 Nov 11.