Bertrand A, Jourdan J, Milane J
Ann Anesthesiol Fr. 1975;16 Spec No 2-3:113-23.
Diffuse pulmonary edema, capable of arising in the absence of hemodynamic disorders is rare in infectious disease. They take on two different clinical appearances: a) acute edema of the lung with the syndrome of asphysia, b) a subacute dyspneic pneumonia with hypoxemia and hypo or normocapnia. These initial disorders can be followed by progressive respiratory failure secondary to the development of diffuse interstitial lesions with fibrosis and intra-alveolar hyaline deposits. The bronchiolo-alveolar lesions which induce a fibrin rich exudate are directly caused by the patogenic agent: myxovirus, essentially influenzae, and more rarely adeno or herpes virus. The role of bacteria and of certain parasites is more debateable.
弥漫性肺水肿在无血流动力学紊乱的情况下发生于传染病中较为罕见。它们呈现出两种不同的临床表现:a)伴有窒息综合征的急性肺水肿,b)伴有低氧血症和低碳酸血症或正常碳酸血症的亚急性呼吸困难性肺炎。这些初始病症之后可能会继发于弥漫性间质性病变伴纤维化和肺泡内透明膜沉积而导致的进行性呼吸衰竭。引起富含纤维蛋白渗出物的细支气管肺泡病变直接由病原体引起:黏液病毒,主要是流感病毒,较少见的是腺病毒或疱疹病毒。细菌和某些寄生虫的作用更具争议性。