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[肺水肿。分类与机制]

[Pulmonary edemas. Classification and mechanisms].

作者信息

Feiss P, Gay R

出版信息

Ann Anesthesiol Fr. 1975;16 Spec No 2-3:11-6.

PMID:9853
Abstract

Water exchanges in the lung under physiological conditions, are still badly known. Starling's classical equation is a too simplified view of the phenomenon. In addition, a certain number of its factors are badly determined. Such is the case with the hydrostatic pressure and the oncotic pressure of the interstitium. In pathological conditions secondary edema is opposed to lesions of the alveolocapillary membrane and hemodynamic edema where the increase in intravascular pressure increases a loss of liquid between the junctions of the endothelial cells. Between these two situations there are edemas, of which the mechanism is still subject to discussion, for example altitude edema, edema in heroin addicts, neurogenic edema.

摘要

在生理条件下,肺内的水交换情况仍鲜为人知。斯塔林的经典方程对该现象的描述过于简化。此外,其若干因素的测定并不准确。肺间质静水压和胶体渗透压的情况便是如此。在病理状态下,继发性水肿与肺泡毛细血管膜病变相对,而血流动力学性水肿是血管内压力升高导致内皮细胞连接处液体流失增加所致。在这两种情况之间,存在一些机制仍有待探讨的水肿,例如高原性水肿、海洛因成瘾者的水肿、神经源性水肿。

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