拟南芥维管组织中AtPIN1对生长素极性运输的调控

Regulation of polar auxin transport by AtPIN1 in Arabidopsis vascular tissue.

作者信息

Gälweiler L, Guan C, Müller A, Wisman E, Mendgen K, Yephremov A, Palme K

机构信息

Max-Delbrück-Laboratorium in der Max-Planck-Gesellschaft, Carl-von-Linné-Weg 10, D-50829 Köln, Germany.

出版信息

Science. 1998 Dec 18;282(5397):2226-30. doi: 10.1126/science.282.5397.2226.

DOI:10.1126/science.282.5397.2226

PMID:9856939

Abstract

Polar auxin transport controls multiple developmental processes in plants, including the formation of vascular tissue. Mutations affecting the PIN-FORMED (PIN1) gene diminish polar auxin transport in Arabidopsis thaliana inflorescence axes. The AtPIN1gene was found to encode a 67-kilodalton protein with similarity to bacterial and eukaryotic carrier proteins, and the AtPIN1 protein was detected at the basal end of auxin transport-competent cells in vascular tissue. AtPIN1 may act as a transmembrane component of the auxin efflux carrier.

摘要

生长素极性运输控制植物的多个发育过程，包括维管组织的形成。影响PIN形成蛋白（PIN1）基因的突变会减少拟南芥花序轴中的生长素极性运输。人们发现AtPIN1基因编码一种67千道尔顿的蛋白质，与细菌和真核载体蛋白相似，并且在维管组织中具备生长素运输能力的细胞的基部末端检测到了AtPIN1蛋白。AtPIN1可能作为生长素流出载体的跨膜组分发挥作用。