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The plasma kallikrein-kinin system in sepsis, inflammatory arthritis, and enterocolitis.

作者信息

Colman R W, Sartor R B, Adam A A, DeLa Cadena R A, Stadnicki A

机构信息

Sol Sherry Thrombosis Research Center, Temple University School of Medicine, Philadelphia, PA 19140, USA.

出版信息

Clin Rev Allergy Immunol. 1998 Winter;16(4):365-84. doi: 10.1007/BF02737657.

DOI:10.1007/BF02737657
PMID:9926286
Abstract
摘要

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Clin Rev Allergy Immunol. 1998 Winter;16(4):365-84. doi: 10.1007/BF02737657.
2
Selective kallikrein-kinin system activation in inbred rats differentially susceptible to granulomatous enterocolitis.在对肉芽肿性小肠结肠炎易感性不同的近交系大鼠中，选择性激肽释放酶-激肽系统激活。
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3
[An experimental model of depletion of the blood kallikrein-kinin system].[血液激肽释放酶-激肽系统耗竭的实验模型]
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The Plasma Kallikrein-Kininogen Pathway Is Critical in the Pathogenesis of Colitis in Mice.血浆激肽释放酶-激肽原系统在小鼠结肠炎发病机制中起关键作用。
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本文引用的文献

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Stimulation of human neutrophil leukocyte aerobic glucose metabolism by purified chemotactic factors.纯化趋化因子对人中性粒细胞有氧葡萄糖代谢的刺激作用。
J Clin Invest. 1974 Feb;53(2):591-9. doi: 10.1172/JCI107594.
2
Kallikrein-kininogen system activation and bradykinin (B2) receptors in indomethacin induced enterocolitis in genetically susceptible Lewis rats.在基因易感性Lewis大鼠中，激肽释放酶-激肽原系统激活及缓激肽（B2）受体与吲哚美辛诱导的小肠结肠炎
Gut. 1998 Sep;43(3):365-74. doi: 10.1136/gut.43.3.365.
3
Localization and secretion of tissue kallikrein in peptidoglycan-induced enterocolitis in Lewis rats.
激肽释放酶-激肽系统在神经紊乱中的意义：探寻潜在的生物标志物和机制。
Prog Neurobiol. 2018 Jun-Aug;165-167:26-50. doi: 10.1016/j.pneurobio.2018.01.003. Epub 2018 Jan 31.
4
Contact pathway of coagulation and inflammation.凝血和炎症的接触途径。
Thromb J. 2015 May 6;13:17. doi: 10.1186/s12959-015-0048-y. eCollection 2015.
5
Kallikreins 5, 6 and 10 differentially alter pathophysiology and overall survival in an ovarian cancer xenograft model.激肽释放酶 5、6 和 10 差异改变卵巢癌异种移植模型中的病理生理学和总生存期。
PLoS One. 2011;6(11):e26075. doi: 10.1371/journal.pone.0026075. Epub 2011 Nov 15.
6
Cleaved high-molecular-weight kininogen and its domain 5 inhibit migration and invasion of human prostate cancer cells through the epidermal growth factor receptor pathway.裂解的高分子量激肽原及其结构域5通过表皮生长因子受体途径抑制人前列腺癌细胞的迁移和侵袭。
Oncogene. 2009 Jul 30;28(30):2756-65. doi: 10.1038/onc.2009.132. Epub 2009 Jun 1.
7
Ferritin binds to light chain of human H-kininogen and inhibits kallikrein-mediated bradykinin release.铁蛋白与人H-激肽原轻链结合并抑制激肽释放酶介导的缓激肽释放。
Biochem J. 2002 Jul 1;365(Pt 1):279-86. doi: 10.1042/BJ20011637.
组织激肽释放酶在肽聚糖诱导的Lewis大鼠小肠结肠炎中的定位与分泌
Am J Physiol. 1998 Oct;275(4):G854-61. doi: 10.1152/ajpgi.1998.275.4.G854.
4
Recombinant tumor necrosis factor receptor p75 fusion protein (TNFR:Fc) alters endotoxin-induced activation of the kinin, fibrinolytic, and coagulation systems in normal humans.重组肿瘤坏死因子受体p75融合蛋白（TNFR:Fc）可改变内毒素诱导的正常人激肽、纤溶和凝血系统的激活。
Thromb Haemost. 1998 Jul;80(1):114-8.
5
Specific inhibition of plasma kallikrein modulates chronic granulomatous intestinal and systemic inflammation in genetically susceptible rats.血浆激肽释放酶的特异性抑制可调节遗传易感大鼠的慢性肉芽肿性肠道和全身炎症。
FASEB J. 1998 Mar;12(3):325-33. doi: 10.1096/fasebj.12.3.325.
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Involvement of endogenous kinins in the pathogenesis of peptidoglycan-induced arthritis in the Lewis rat.内源性激肽在Lewis大鼠肽聚糖诱导的关节炎发病机制中的作用。
Arthritis Rheum. 1997 Jul;40(7):1327-33. doi: 10.1002/1529-0131(199707)40:7<1327::AID-ART18>3.0.CO;2-B.
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Current concepts of the etiology and pathogenesis of ulcerative colitis and Crohn's disease.溃疡性结肠炎和克罗恩病的病因及发病机制的当前概念
Gastroenterol Clin North Am. 1995 Sep;24(3):475-507.
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Inhibition of factor XII in septic baboons attenuates the activation of complement and fibrinolytic systems and reduces the release of interleukin-6 and neutrophil elastase.抑制脓毒症狒狒中的因子XII可减弱补体和纤溶系统的激活，并减少白细胞介素-6和中性粒细胞弹性蛋白酶的释放。
Blood. 1996 Mar 15;87(6):2337-44.
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Selective plasma kallikrein inhibitor attenuates acute intestinal inflammation in Lewis rat.选择性血浆激肽释放酶抑制剂可减轻Lewis大鼠的急性肠道炎症。
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10
Selective kallikrein-kinin system activation in inbred rats differentially susceptible to granulomatous enterocolitis.在对肉芽肿性小肠结肠炎易感性不同的近交系大鼠中，选择性激肽释放酶-激肽系统激活。
Gastroenterology. 1996 May;110(5):1467-81. doi: 10.1053/gast.1996.v110.pm8613052.