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骨佩吉特病和原发性甲状旁腺功能亢进症中尿羟赖氨酸糖苷的来源。

The origin of urinary hydroxylysyl glycosides in Paget's disease of bone and in primary hyperparathyroidism.

作者信息

Askenasi R, De Backer M, Devos A

出版信息

Calcif Tissue Res. 1976 Nov 24;22(1):35-40. doi: 10.1007/BF02010344.

DOI:10.1007/BF02010344
PMID:1000341
Abstract

The urinary excretion of glucosyl-galactosyl-hydroxylysine and of galactosyl-hydroxylysine were studied in Paget's disease of bone and in primary hyperparathyroidism. Both metabolites were increased in these diseases. Although glucosyl-galactosyl-hydroxylysine is not abundant in bone collagen, it is possible that a part of it originates from calcified tissue.

摘要

对骨Paget病和原发性甲状旁腺功能亢进症患者的葡萄糖基 - 半乳糖基 - 羟赖氨酸及半乳糖基 - 羟赖氨酸的尿排泄情况进行了研究。在这些疾病中,这两种代谢产物均有所增加。尽管葡萄糖基 - 半乳糖基 - 羟赖氨酸在骨胶原中含量并不丰富,但它有可能部分来源于钙化组织。

相似文献

1
The origin of urinary hydroxylysyl glycosides in Paget's disease of bone and in primary hyperparathyroidism.骨佩吉特病和原发性甲状旁腺功能亢进症中尿羟赖氨酸糖苷的来源。
Calcif Tissue Res. 1976 Nov 24;22(1):35-40. doi: 10.1007/BF02010344.
2
Urinary excretion of hydroxylysine and its glycosides as an index of collagen degradation.以羟赖氨酸及其糖苷的尿排泄量作为胶原蛋白降解的指标。
J Clin Invest. 1977 May;59(5):819-27. doi: 10.1172/JCI108704.
3
Urinary excretion of hydroxyproline, hydroxylysine and hydroxylysine glycosides by patients with Paget's disease of bone and carcinoma with metastases in bone.患有骨Paget病及骨转移癌的患者尿中羟脯氨酸、羟赖氨酸及羟赖氨酸糖苷的排泄情况。
Clin Chim Acta. 1979 Mar 15;92(3):373-9. doi: 10.1016/0009-8981(79)90216-x.
4
Urinary hydroxylysine and hydroxylysyl glycoside excretions in normal and pathologic states.正常及病理状态下尿中羟赖氨酸及羟赖氨酰糖苷的排泄情况。
J Lab Clin Med. 1974 Apr;83(4):673-9.
5
Urinary excretion of hydroxylysyl glycosides and thyroid function.
J Clin Endocrinol Metab. 1975 Feb;40(2):342-4. doi: 10.1210/jcem-40-2-342.
6
Contribution of kidney basement membranes and collagen to the urinary excretion of hydroxylysyl glycosides.
Biomedicine. 1975 May;22(3):233-6.
7
Urinary excretion of free hydroxylysine, peptide-bound hydroxylysine and hydroxylysyl glycosides in physiological conditions.
Clin Chim Acta. 1975 Feb 22;59(1):87-92. doi: 10.1016/0009-8981(75)90222-3.
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Urinary excretion of pyridinium crosslinks: a new marker of bone resorption in metabolic bone disease.吡啶交联物的尿排泄:代谢性骨病中骨吸收的新标志物。
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Urinary excretion of hydroxylysyl glycosides during acute inflammation in the rat.
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Galactosyl hydroxylysine in assessment of Paget's bone disease.
Clin Chim Acta. 1995 Jan 31;234(1-2):101-8. doi: 10.1016/0009-8981(94)05981-w.

本文引用的文献

1
Hydroxylysine glycosides in human urine.人尿中的羟赖氨酸糖苷
Clin Chem. 1971 Aug;17(8):782-8.
2
Radioimmunoassay of human parathyroid hormone in serum.血清中人类甲状旁腺激素的放射免疫测定
J Clin Invest. 1971 Jan;50(1):21-34. doi: 10.1172/JCI106476.
3
Variations in human urinary O-hydroxylysyl glycoside levels and their relationship to collagen metabolism.人类尿液中O-羟赖氨酸糖苷水平的变化及其与胶原蛋白代谢的关系。
J Clin Invest. 1970 Aug;49(8):1497-509. doi: 10.1172/JCI106367.
4
Human collagens: differences in glycosylated hydroxylysines in skin and bone.人类胶原蛋白:皮肤和骨骼中糖基化羟赖氨酸的差异。
Biochim Biophys Acta. 1971 Jan 19;229(1):119-22. doi: 10.1016/0005-2795(71)90325-4.
5
A heritable disorder of connective tissue. Hydroxylysine-deficient collagen disease.一种遗传性结缔组织疾病。羟赖氨酸缺乏性胶原病。
N Engl J Med. 1972 May 11;286(19):1013-20. doi: 10.1056/NEJM197205112861901.
6
Urinary hydroxylysine and hydroxylysyl glycoside excretions in normal and pathologic states.正常及病理状态下尿中羟赖氨酸及羟赖氨酰糖苷的排泄情况。
J Lab Clin Med. 1974 Apr;83(4):673-9.
7
A new rapid method for measuring hydroxylysine and its glycosides in hydrolysates and physiological fluids.一种用于测量水解产物和生理流体中羟赖氨酸及其糖苷的新快速方法。
Biochim Biophys Acta. 1973 Apr 28;304(2):375-83. doi: 10.1016/0304-4165(73)90256-0.
8
C1q protein of human complement.人类补体C1q蛋白
Biochemistry. 1972 Aug 29;11(18):3443-50. doi: 10.1021/bi00768a018.
9
A protein binding assay for adenosine 3':5'-cyclic monophosphate.一种用于检测3':5'-环磷酸腺苷的蛋白质结合测定法。
Proc Natl Acad Sci U S A. 1970 Sep;67(1):305-12. doi: 10.1073/pnas.67.1.305.
10
Urinary polypeptides related to collagen synthesis.与胶原蛋白合成相关的尿多肽。
J Clin Invest. 1970 Apr;49(4):716-29. doi: 10.1172/JCI106284.