Pagán R, Mañas P, Palop A, Sala F J
Tecnología de los Alimentos, Facultad de Veterinaria, Universidad de Zaragoza, Spain.
Lett Appl Microbiol. 1999 Jan;28(1):71-5. doi: 10.1046/j.1365-2672.1999.00465.x.
Heat shocks did not increase the resistance of Listeria monocytogenes to an ultrasonication treatment under pressure (Mano-Sonication; MS). While heat-shocked cells (180 min, 45 degrees C) became sixfold more heat resistant than native cells (D62 = 1.8 min vs D62 = 0.24 min), the resistance of native and heat-shocked cells to MS (200 kPa, 117 microns) was the same (DMS = 1.6 min). The inactivation rate of non-heat-shocked cells of L monocytogenes by a combined heat/ultrasonication treatment under pressure (Mano-Thermo-Sonication; MTS) was an additive effect. On the contrary, on heat-shocked cells, the inactivation rate of MTS was greater than that of heat added to the inactivation rate of MS (synergistic effect) in the range 62-68 degrees C.
热休克并未增加单核细胞增生李斯特菌对压力下超声处理(压力辅助超声处理;MS)的抗性。虽然热休克细胞(180分钟,45摄氏度)的耐热性比天然细胞提高了六倍(D62 = 1.8分钟对D62 = 0.24分钟),但天然细胞和热休克细胞对MS(200千帕,117微米)的抗性相同(DMS = 1.6分钟)。压力下热/超声联合处理(压力辅助热超声处理;MTS)对非热休克单核细胞增生李斯特菌的失活率具有累加效应。相反,在62 - 68摄氏度范围内,对于热休克细胞,MTS的失活率大于热与MS失活率之和(协同效应)。