Ono K, Takizawa Y, Komai K, Takamori M
Department of Neurology, Kanazawa University School of Medicine.
Rinsho Shinkeigaku. 1998 Sep;38(9):806-10.
A 63-year-old woman suddenly began to suffer from left chest pain. She gradually became unable to walk and was admitted to the emergency room at another hospital. When she became paraplegic in spite of steroid therapy, she was admitted to our hospital. Her affliction was diagnosed as anterior spinal artery syndrome because of flaccid paraplegia and dissociated sensory loss below the Th4 dermatome. Hematological study indicated a compensated DIC and hepatic enzyme abnormality, while the CSF examinations showed an elevation of protein and positive myelin basic protein (MBP) elevation. The initial MRI taken in the acute stage showed no abnormal signals on T1-weighted (T1) and Gd-enhanced images. The sagittal T2-weighted image (T2) revealed central high intensity (HI) with longitudinal extension from Th2 through the Th11 vertebral level. On axial T2, HI was located on the gray matter at the Th3 and Th4 vertebral level, the ventral two-thirds at the Th8 vertebral level, the central ventral side at the Th9 and Th10 vertebral level, and the entire cross section at the Th12 and L1. A follow-up MRI examination showed that the range of HI on the sagittal T2 had been reduced to 5 segments from Th6 through Th10 vertebral level. The T2 HI lesion on the axial aspect had become reduced so as to localize on the left ventral side at the Th8 vertebral level and on the central ventral side at Th9 and Th10.
一名63岁女性突然开始出现左胸痛。她逐渐无法行走,被送往另一家医院的急诊室。尽管接受了类固醇治疗,但她仍出现截瘫,随后被收治入我院。由于弛缓性截瘫和T4皮节以下感觉分离性丧失,她的病情被诊断为脊髓前动脉综合征。血液学检查显示存在代偿性弥散性血管内凝血(DIC)和肝酶异常,而脑脊液检查显示蛋白升高且髓鞘碱性蛋白(MBP)升高呈阳性。急性期的初次磁共振成像(MRI)在T1加权(T1)和钆增强图像上未显示异常信号。矢状位T2加权图像(T2)显示中央高强度(HI),从T2至T11椎体水平呈纵向延伸。在轴位T2上,HI位于T3和T4椎体水平的灰质、T8椎体水平的腹侧三分之二、T9和T10椎体水平的中央腹侧以及T12和L1的整个横截面。随访MRI检查显示,矢状位T2上HI的范围已从T6至T10椎体水平缩小至5个节段。轴位方面的T2 HI病变已缩小,局限于T8椎体水平的左侧腹侧以及T9和T10椎体水平的中央腹侧。