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1
Immunomodulation by interference with co-stimulatory molecules: therapeutic perspectives in asthma.通过干扰共刺激分子进行免疫调节:哮喘的治疗前景
Thorax. 1999 Jun;54(6):554-7. doi: 10.1136/thx.54.6.554.
2
Dendritic cells can be used as physiological adjuvant in vivo.树突状细胞可在体内用作生理性佐剂。
Adv Exp Med Biol. 1995;378:501-5. doi: 10.1007/978-1-4615-1971-3_113.
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Expression of B7 costimulator molecules on mouse dendritic cells.小鼠树突状细胞上B7共刺激分子的表达
Adv Exp Med Biol. 1995;378:65-70. doi: 10.1007/978-1-4615-1971-3_13.
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Adv Exp Med Biol. 1997;417:203-6. doi: 10.1007/978-1-4757-9966-8_34.
5
Improved isolation of dendritic cells in chronic arthritic joints reveals no B7 (CD80) surface expression.慢性关节炎关节中树突状细胞分离方法的改进显示其无B7(CD80)表面表达。
Adv Exp Med Biol. 1995;378:561-3. doi: 10.1007/978-1-4615-1971-3_126.
6
Phenotype and allostimulatory function of dendritic cells treated with antisense oligodeoxyribonucleotides targeting CD80 or CD86 mRNA.用靶向CD80或CD86 mRNA的反义寡脱氧核糖核苷酸处理的树突状细胞的表型和共刺激功能。
Transplant Proc. 2001 Feb-Mar;33(1-2):235. doi: 10.1016/s0041-1345(00)01991-6.
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The role of dendritic cells in T cell activation.树突状细胞在T细胞活化中的作用。
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Immunophenotypical and functional characterization of bone marrow derived dendritic cells.骨髓来源树突状细胞的免疫表型和功能特征
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B7 costimulation plays an important role in protection from herpes simplex virus type 2-mediated pathology.B7共刺激在抵御2型单纯疱疹病毒介导的病理过程中发挥重要作用。
J Virol. 2002 Mar;76(5):2563-6. doi: 10.1128/jvi.76.5.2563-2566.2002.

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J Immunol. 2003 Aug 15;171(4):1676-83. doi: 10.4049/jimmunol.171.4.1676.
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Novel drugs for treating asthma.
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本文引用的文献

1
Costimulation through CD86 is involved in airway antigen-presenting cell and T cell responses to allergen in atopic asthmatics.通过CD86的共刺激参与了特应性哮喘患者气道抗原呈递细胞和T细胞对变应原的反应。
J Immunol. 1998 Dec 1;161(11):6375-82.
2
Selective differences in the expression of the homing receptors of helper lymphocyte subsets.辅助淋巴细胞亚群归巢受体表达的选择性差异。
Clin Immunol Immunopathol. 1998 Nov;89(2):110-6. doi: 10.1006/clin.1998.4589.
3
Studies using antigen-presenting cells lacking expression of both B7-1 (CD80) and B7-2 (CD86) show distinct requirements for B7 molecules during priming versus restimulation of Th2 but not Th1 cytokine production.使用缺乏B7-1(CD80)和B7-2(CD86)表达的抗原呈递细胞进行的研究表明,在Th2细胞因子产生的启动与再刺激过程中,对B7分子有不同的需求,但对Th1细胞因子产生则不然。
J Immunol. 1998 Sep 15;161(6):2762-71.
4
Delivery of salbutamol to nonventilated preterm infants by metered-dose inhaler, jet nebulizer, and ultrasonic nebulizer.通过定量吸入器、喷射雾化器和超声雾化器向未通气的早产儿递送沙丁胺醇。
Eur Respir J. 1998 Jul;12(1):159-64. doi: 10.1183/09031936.98.12010159.
5
CD40 and CD40 ligand (CD154) are coexpressed on microvessels in vivo in human cardiac allograft rejection.在人类心脏同种异体移植排斥反应中,CD40和CD40配体(CD154)在体内微血管上共同表达。
Transplantation. 1997 Dec 27;64(12):1765-74. doi: 10.1097/00007890-199712270-00025.
6
Recruitment of circulating allergen-specific T lymphocytes to the lung on allergen challenge in asthma.在哮喘患者中,变应原激发时循环中的变应原特异性T淋巴细胞向肺部募集。
J Allergy Clin Immunol. 1997 Nov;100(5):669-78. doi: 10.1016/s0091-6749(97)70172-6.
7
Murine CTLA4-IgG treatment inhibits airway eosinophilia and hyperresponsiveness and attenuates IgE upregulation in a murine model of allergic asthma.在过敏性哮喘小鼠模型中,鼠源CTLA4-IgG治疗可抑制气道嗜酸性粒细胞增多和高反应性,并减弱IgE上调。
Am J Respir Cell Mol Biol. 1997 Sep;17(3):386-92. doi: 10.1165/ajrcmb.17.3.2679.
8
Differential regulation of human, antigen-specific Th1 and Th2 responses by the B-7 homologues, CD80 and CD86.B-7同源物CD80和CD86对人抗原特异性Th1和Th2应答的差异调节
Am J Respir Cell Mol Biol. 1997 Aug;17(2):235-42. doi: 10.1165/ajrcmb.17.2.2739.
9
CTLA4-Ig and anti-CD40 ligand prevent renal allograft rejection in primates.CTLA4免疫球蛋白和抗CD40配体可预防灵长类动物的肾移植排斥反应。
Proc Natl Acad Sci U S A. 1997 Aug 5;94(16):8789-94. doi: 10.1073/pnas.94.16.8789.
10
Alveolar macrophages in sarcoidosis coexpress high levels of CD86 (B7.2), CD40, and CD30L.结节病中的肺泡巨噬细胞共表达高水平的CD86(B7.2)、CD40和CD30L。
Am J Respir Cell Mol Biol. 1997 Jul;17(1):91-6. doi: 10.1165/ajrcmb.17.1.2781.

Immunomodulation by interference with co-stimulatory molecules: therapeutic perspectives in asthma.

作者信息

Burastero S E, Rossi G A

机构信息

San Raffaele Institute, 20132 Milan, Italy.

出版信息

Thorax. 1999 Jun;54(6):554-7. doi: 10.1136/thx.54.6.554.

DOI:10.1136/thx.54.6.554
PMID:10335013
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC1745485/
Abstract
摘要