Shirai S, Tomono Y, Owada T, Maki Y
No Shinkei Geka. 1976 Jun;4(6):605-10.
Three cases of traumatic thrombosis of the internal carotid artery due to head injury with basal skull fracture were reported. The mechanism of thrombosis, the symptomatologic characteristics and the prognosis were discussed with reference to the data in the literature. Four mechanisms by which the condition may be produced include; injury to the intrapetrous or cevernous portion of the carotid artery during basal skull fracture, injury to the point of emergence of the carotid artery from the cavernous sinus as a result of shearing strains suffered at the time of injury, a direct blow to the neck or trauma to the paratonsillar area by a foreign object carried in the mouth, and stretching of the carotid artery by heperextension and lateral flexion of the neck. Secondary symptoms due to thrombosis of the carotid artery after an interval develop most commonly from twelve to twenty four hours. The prognosis in these patients is mostly poor. Therefore, angiography should be performed immediately on any patient suspected of having this lesion and treatment should be considered.
报告了3例因头部受伤合并颅底骨折导致颈内动脉创伤性血栓形成的病例。结合文献资料对血栓形成的机制、症状学特征及预后进行了讨论。该病可能的产生机制有四种:颅底骨折时岩骨内或海绵窦段颈内动脉损伤;受伤时因剪切力导致颈动脉从海绵窦穿出点损伤;颈部直接受击或口腔内异物对扁桃体旁区域造成创伤;颈部过度伸展和侧屈导致颈动脉拉伸。颈动脉血栓形成后的继发症状最常在12至24小时后出现。这些患者的预后大多较差。因此,对于任何疑似患有此病变的患者应立即进行血管造影,并考虑进行治疗。