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剪接元件和聚腺苷酸化信号元件在聚腺苷酸化与最后一个内含子去除偶联中的利用

Utilization of splicing elements and polyadenylation signal elements in the coupling of polyadenylation and last-intron removal.

作者信息

Cooke C, Hans H, Alwine J C

机构信息

Department of Microbiology, School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania 19104-6142, USA.

出版信息

Mol Cell Biol. 1999 Jul;19(7):4971-9. doi: 10.1128/MCB.19.7.4971.

Abstract

Polyadenylation (PA) is the process by which the 3' ends of most mammalian mRNAs are formed. In nature, PA is highly coordinated, or coupled, with splicing. In mammalian systems, the most compelling mechanistic model for coupling arises from data supporting exon definition (2, 34, 37). We have examined the roles of individual functional components of splicing and PA signals in the coupling process by using an in vitro splicing and PA reaction with a synthetic pre-mRNA substrate containing an adenovirus splicing cassette and the simian virus 40 late PA signal. The effects of individually mutating splicing elements and PA elements in this substrate were determined. We found that mutation of the polypyrimidine tract and the 3' splice site significantly reduced PA efficiency and that mutation of the AAUAAA and the downstream elements of the PA signal decreased splicing efficiency, suggesting that these elements are the most significant for the coupling of splicing and PA. Although mutation of the upstream elements (USEs) of the PA signal dramatically decreased PA, splicing was only modestly affected, suggesting that USEs modestly affect coupling. Mutation of the 5' splice site in the presence of a viable polypyrimidine tract and the 3' splice site had no effect on PA, suggesting no effect of this element on coupling. However, our data also suggest that a site for U1 snRNP binding (e.g., a 5' splice site) within the last exon can negatively effect both PA and splicing; hence, a 5' splice site-like sequence in this position appears to be a modulator of coupling. In addition, we show that the RNA-protein complex formed to define an exon may inhibit processing if the definition of an adjacent exon fails. This finding indicates a mechanism for monitoring the appropriate definition of exons and for allowing only pre-mRNAs with successfully defined exons to be processed.

摘要

聚腺苷酸化(PA)是大多数哺乳动物mRNA 3'末端形成的过程。在自然界中,PA与剪接高度协调或偶联。在哺乳动物系统中,支持外显子定义的数据(2, 34, 37)产生了最有说服力的偶联机制模型。我们通过使用含有腺病毒剪接盒和猿猴病毒40晚期PA信号的合成前体mRNA底物进行体外剪接和PA反应,研究了剪接和PA信号的各个功能成分在偶联过程中的作用。确定了该底物中剪接元件和PA元件单独突变的影响。我们发现,多嘧啶序列和3'剪接位点的突变显著降低了PA效率,而PA信号的AAUAAA和下游元件的突变降低了剪接效率,这表明这些元件对剪接和PA的偶联最为重要。虽然PA信号上游元件(USEs)的突变显著降低了PA,但剪接仅受到适度影响,表明USEs对偶联有适度影响。在存在可行的多嘧啶序列和3'剪接位点的情况下,5'剪接位点的突变对PA没有影响,表明该元件对偶联没有影响。然而,我们的数据还表明,最后一个外显子内的U1 snRNP结合位点(例如5'剪接位点)可对PA和剪接产生负面影响;因此,该位置的5'剪接位点样序列似乎是偶联的调节因子。此外,我们表明,如果相邻外显子的定义失败,为定义外显子而形成的RNA-蛋白质复合物可能会抑制加工。这一发现揭示了一种监测外显子适当定义并仅允许具有成功定义外显子的前体mRNA进行加工的机制。

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