Tumour necrosis factor and other cytokines in murine lupus.
作者信息
Theofilopoulos A N, Lawson B R
机构信息
Department of Immunology, The Scripps Research Institute, La Jolla, USA.
出版信息
Ann Rheum Dis. 1999 Nov;58 Suppl 1(Suppl 1):I49-55. doi: 10.1136/ard.58.2008.i49.
Abstract
摘要
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本文引用的文献
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Modulation of autoimmune disease in the MRL-lpr/lpr mouse by IL-2 and TGF-beta1 gene therapy using attenuated Salmonella typhimurium as gene carrier.使用减毒鼠伤寒沙门氏菌作为基因载体，通过白细胞介素-2和转化生长因子-β1基因疗法对MRL-lpr/lpr小鼠自身免疫性疾病的调节。
Lupus. 1999;8(1):29-38. doi: 10.1191/096120399678847308.
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Requirement for transcription factor NFAT in interleukin-2 expression.白细胞介素-2表达中对转录因子NFAT的需求。
Mol Cell Biol. 1999 Mar;19(3):2300-7. doi: 10.1128/MCB.19.3.2300.
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Regulation of autoimmunity by proinflammatory cytokines.促炎细胞因子对自身免疫的调节
Curr Opin Immunol. 1998 Dec;10(6):669-76. doi: 10.1016/s0952-7915(98)80087-3.
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Intrinsic defects in macrophage IL-12 production associated with immune dysfunction in the MRL/++ and New Zealand Black/White F1 lupus-prone mice and the Leishmania major-susceptible BALB/c strain.MRL/++和新西兰黑/白F1狼疮易感小鼠以及利什曼原虫主要易感BALB/c品系中，巨噬细胞白细胞介素-12产生的内在缺陷与免疫功能障碍相关。
J Immunol. 1998 Dec 15;161(12):6878-84.
5
c-maf promotes T helper cell type 2 (Th2) and attenuates Th1 differentiation by both interleukin 4-dependent and -independent mechanisms.c-Maf通过依赖白细胞介素4和不依赖白细胞介素4的机制促进2型辅助性T细胞（Th2）分化并减弱Th1分化。
J Exp Med. 1998 Nov 16;188(10):1859-66. doi: 10.1084/jem.188.10.1859.
6
Dissection of the effects of tumor necrosis factor-alpha and class II gene polymorphisms within the MHC on murine systemic lupus erythematosus (SLE).剖析肿瘤坏死因子-α及主要组织相容性复合体（MHC）内II类基因多态性对小鼠系统性红斑狼疮（SLE）的影响。
Int Immunol. 1998 Oct;10(10):1467-72. doi: 10.1093/intimm/10.10.1467.
7
Feedback inhibition of macrophage tumor necrosis factor-alpha production by tristetraprolin.Tristetraprolin对巨噬细胞肿瘤坏死因子-α产生的反馈抑制作用。
Science. 1998 Aug 14;281(5379):1001-5. doi: 10.1126/science.281.5379.1001.
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Atypical signaling defects prevent IL-2 gene expression in lpr/lpr CD4-CD8- cells.非典型信号缺陷阻止lpr/lpr CD4-CD8-细胞中白细胞介素-2基因的表达。
J Biomed Sci. 1998 Jul-Aug;5(4):297-304. doi: 10.1007/BF02255862.
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Interferon-gamma expression by Th1 effector T cells mediated by the p38 MAP kinase signaling pathway.由p38丝裂原活化蛋白激酶信号通路介导的Th1效应T细胞的γ干扰素表达。
EMBO J. 1998 May 15;17(10):2817-29. doi: 10.1093/emboj/17.10.2817.
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