Kobayashi Y, Kaya H, Goto K, Iwabuchi M, Araki T
Department of Botany, Graduate School of Science, Kyoto University, Sakyo-ku, Kyoto 606-8502, Japan.
Science. 1999 Dec 3;286(5446):1960-2. doi: 10.1126/science.286.5446.1960.
Flowering in Arabidopsis is promoted via several interacting pathways. A photoperiod-dependent pathway relays signals from photoreceptors to a transcription factor gene, CONSTANS (CO), which activates downstream meristem identity genes such as LEAFY (LFY). FT, together with LFY, promotes flowering and is positively regulated by CO. Loss of FT causes delay in flowering, whereas overexpression of FT results in precocious flowering independent of CO or photoperiod. FT acts in part downstream of CO and mediates signals for flowering in an antagonistic manner with its homologous gene, TERMINAL FLOWER1 (TFL1).
拟南芥的开花通过几种相互作用的途径被促进。一个光周期依赖途径将信号从光感受器传递到一个转录因子基因CONSTANS(CO),该基因激活下游的分生组织特性基因,如LEAFY(LFY)。FT与LFY一起促进开花,并受到CO的正向调控。FT缺失会导致开花延迟,而FT的过表达会导致不依赖CO或光周期的早熟开花。FT部分作用于CO的下游,并与其同源基因TERMINAL FLOWER1(TFL1)以拮抗方式介导开花信号。