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Prolonged reductions in the thresholds of evoked epileptiform discharges in slices of rat hippocampal field CA1 induced by periodic removal of Mg2+.

作者信息

Sem'yanov A V, Godukhin O V

机构信息

Institute of Theoretical and Experimental Biophysics, Russian Academy of Sciences, Moscow Region.

出版信息

Neurosci Behav Physiol. 2000 Mar-Apr;30(2):237-40. doi: 10.1007/BF02463164.

DOI:10.1007/BF02463164
PMID:10872736
Abstract
摘要

相似文献

1
Prolonged reductions in the thresholds of evoked epileptiform discharges in slices of rat hippocampal field CA1 induced by periodic removal of Mg2+.通过周期性去除镁离子诱导大鼠海马CA1区切片中诱发癫痫样放电阈值的长期降低。
Neurosci Behav Physiol. 2000 Mar-Apr;30(2):237-40. doi: 10.1007/BF02463164.
2
Chloride-cotransport blockade desynchronizes neuronal discharge in the "epileptic" hippocampal slice.氯离子共转运体阻断使“癫痫性”海马切片中的神经元放电不同步。
J Neurophysiol. 2000 Jan;83(1):406-17. doi: 10.1152/jn.2000.83.1.406.
3
Low-magnesium epilepsy in rat hippocampal slices: inhibitory postsynaptic potentials in the CA1 subfield.大鼠海马切片中的低镁癫痫：CA1 亚区的抑制性突触后电位
Neurosci Lett. 1988 Jul 8;89(3):293-8. doi: 10.1016/0304-3940(88)90542-3.
4
Increases in the threshold for the development of epileptiform activity in field CA1 of Krushinskii-Molodnika rat hippocampal slices as an adaptive protective mechanism.作为一种适应性保护机制，克鲁申斯基-莫洛德尼卡大鼠海马切片CA1区癫痫样活动发展阈值的升高。
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5
Epileptiform activity and changes in field potential responses induced by low [Mg2+]0 in a genetic rat model of absence epilepsy.失神癫痫遗传大鼠模型中低细胞外镁离子浓度诱导的癫痫样活动及场电位反应变化
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6
Effects of variations in hippocampal slice preparation protocol on the electrophysiological stability, epileptogenicity and graded hypoxia responses of CA1 neurons.海马切片制备方案的变化对CA1神经元电生理稳定性、致痫性和分级缺氧反应的影响。
Brain Res. 1997 Nov 14;775(1-2):134-43. doi: 10.1016/s0006-8993(97)00893-7.
7
Reduced Mg2+ blockade of synaptically activated N-methyl-D-aspartate receptor-channels in CA1 pyramidal neurons in kainic acid-lesioned rat hippocampus.在 kainic 酸损伤的大鼠海马体 CA1 锥体神经元中，突触激活的 N-甲基-D-天冬氨酸受体通道的 Mg2+ 阻断作用减弱。
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8
Effects of the triazole derivative loreclezole (R72063) on stimulus induced ionic and field potential responses and on different patterns of epileptiform activity induced by low magnesium in rat entorhinal cortex-hippocampal slices.三唑衍生物洛雷唑（R72063）对大鼠内嗅皮层-海马切片中刺激诱发的离子和场电位反应以及低镁诱导的不同癫痫样活动模式的影响。
Naunyn Schmiedebergs Arch Pharmacol. 1992 Nov;346(5):581-7. doi: 10.1007/BF00169016.
9
Greater contribution of N-methyl-D-aspartic acid receptors in ventral compared to dorsal hippocampal slices in the expression and long-term maintenance of epileptiform activity.与背侧海马切片相比，N-甲基-D-天冬氨酸受体在腹侧海马切片癫痫样活动的表达和长期维持中起更大作用。
Neuroscience. 2005;135(3):765-79. doi: 10.1016/j.neuroscience.2005.06.024. Epub 2005 Sep 9.
10
Synaptic regulation of the slow Ca2+-activated K+ current in hippocampal CA1 pyramidal neurons: implication in epileptogenesis.海马CA1锥体神经元中慢钙激活钾电流的突触调节：对癫痫发生的影响
J Neurophysiol. 2001 Dec;86(6):2878-86. doi: 10.1152/jn.2001.86.6.2878.

本文引用的文献

1
Kindling-like state in rat hippocampal CA1 slices induced by the repeated short-term extracellular K+ increases: the role of L-type Ca(2+)-channels.重复短期细胞外钾离子增加诱导大鼠海马CA1区切片出现类点燃状态：L型钙通道的作用
Neurosci Lett. 1997 Feb 28;223(3):177-80. doi: 10.1016/s0304-3940(97)13436-x.
2
Properties of low Mg2+ induced epileptiform activity in rat hippocampal and entorhinal cortex slices during adolescence.青春期大鼠海马和内嗅皮层切片中低镁离子诱导的癫痫样活动的特性
Brain Res Dev Brain Res. 1995 Jul 14;87(2):145-52. doi: 10.1016/0165-3806(95)00069-p.
3
Magnesium gates glutamate-activated channels in mouse central neurones.
镁离子控制小鼠中枢神经元中谷氨酸激活的通道。
Nature. 1984;307(5950):462-5. doi: 10.1038/307462a0.
4
Magnesium-free medium activates seizure-like events in the rat hippocampal slice.无镁培养基可激活大鼠海马切片中的癫痫样事件。
Brain Res. 1986 Nov 19;398(1):215-9. doi: 10.1016/0006-8993(86)91274-6.
5
How do seizures begin? Clues from hippocampal slices.癫痫发作是如何开始的？来自海马切片的线索。
Trends Neurosci. 1988 Sep;11(9):375-8. doi: 10.1016/0166-2236(88)90070-7.