Brain tissue responses to ischemia.
作者信息
Lee J M, Grabb M C, Zipfel G J, Choi D W
机构信息
Center for the Study of Nervous System Injury and Department of Neurology, Washington University School of Medicine, St. Louis, Missouri 63110, USA.
出版信息
J Clin Invest. 2000 Sep;106(6):723-31. doi: 10.1172/JCI11003.
Abstract
摘要
相似文献
1
Brain tissue responses to ischemia.
J Clin Invest. 2000 Sep;106(6):723-31. doi: 10.1172/JCI11003.
2
Possible mechanisms of pharmacological neuronal protection.
J Neurosurg Anesthesiol. 1995 Jan;7(1):31-7. doi: 10.1097/00008506-199501000-00007.
3
[Brain protection during ischemia: state-of-the-art].
Anesteziol Reanimatol. 2005 Jul-Aug(4):4-14.
4
[Effect of diazoxide on oxygen free radicals and cell apoptosis in brain tissue after deep hypothermia cerebral ischemia reperfusion injury in young rats].
Zhonghua Wai Ke Za Zhi. 2010 Jan 15;48(2):142-5.
5
Is it time to definitely abandon neuroprotection in acute ischemic stroke?
Stroke. 2008 Jun;39(6):1659-60. doi: 10.1161/STROKEAHA.107.505024. Epub 2008 Mar 27.
6
[Cerebral ischemia-hypoxia and biophysical mechanisms of neurodegeneration and neuroprotection effects].
Fiziol Zh (1994). 2003;49(2):7-12.
7
Neuroprotection afforded by prior citicoline administration in experimental brain ischemia: effects on glutamate transport.
Neurobiol Dis. 2005 Mar;18(2):336-45. doi: 10.1016/j.nbd.2004.10.006.
8
Gene expression in cerebral ischemia: a new approach for neuroprotection.
Cerebrovasc Dis. 2006;21 Suppl 2:30-7. doi: 10.1159/000091701. Epub 2006 May 2.
9
[Cerebral ischemia and free radical].
No To Shinkei. 2003 Mar;55(3):201-13.
10
Prevention, treatment, and resuscitation of cerebral ischemia.
Acta Anaesthesiol Scand Suppl. 1997;111:95-9.
引用本文的文献
1
Biological Plausibility of Using Plasma Amino Acid Profile Determination as a Potential Biomarker for Pediatric Patients with Mild Traumatic Brain Injuries.
Neurol Int. 2025 Sep 9;17(9):145. doi: 10.3390/neurolint17090145.
2
Potential impacts of acupuncture on motor function recovery after ischemic stroke: insights from basic and clinical studies.
Front Cell Neurosci. 2025 Aug 13;19:1623535. doi: 10.3389/fncel.2025.1623535. eCollection 2025.
3
Activation of Emergency Department Stroke Protocol by Emergency Medical Services: A Retrospective Cross-Sectional Study.
J Clin Med. 2025 Jul 16;14(14):5041. doi: 10.3390/jcm14145041.
4
Usability of Mixed Reality for Naloxone Training: Iterative Development and Field Testing of ReviveXR.
Healthcare (Basel). 2025 Jun 17;13(12):1449. doi: 10.3390/healthcare13121449.
5
Role of Ischemia/Reperfusion and Oxidative Stress in Shock State.
Cells. 2025 May 30;14(11):808. doi: 10.3390/cells14110808.
6
Neuroserpin alleviates cerebral ischemia-reperfusion injury by suppressing ischemia-induced endoplasmic reticulum stress.
Neural Regen Res. 2026 Jan 1;21(1):333-345. doi: 10.4103/NRR.NRR-D-24-00044. Epub 2024 Sep 6.
7
The role of autophagy in ischemic brain injury.
Autophagy Rep. 2025 Apr 3;4(1):2486445. doi: 10.1080/27694127.2025.2486445. eCollection 2025.
8
Continuous personalized cerebrovascular reactivity-based physiologic metrics in neurocritical care: a narrative review of the current landscape, limitations, and future directions.
Front Physiol. 2025 May 6;16:1582813. doi: 10.3389/fphys.2025.1582813. eCollection 2025.
9
Therapeutic Hypothermia Increases the Expression of RNA-binding Protein Motif 3 and Attenuates Cognitive Deficits Following Cardiac Arrest in Rats.
Neurochem Res. 2025 Apr 3;50(2):134. doi: 10.1007/s11064-025-04383-1.
10
Lesion network mapping of REM Sleep Behaviour Disorder.
Neuroimage Clin. 2025;45:103751. doi: 10.1016/j.nicl.2025.103751. Epub 2025 Feb 7.
本文引用的文献
1
The search for physiological substrates of MAP and SAP kinases in mammalian cells.
Trends Cell Biol. 1997 Sep;7(9):353-61. doi: 10.1016/S0962-8924(97)01105-7.
2
Zn2+ current is mediated by voltage-gated Ca2+ channels and enhanced by extracellular acidity in mouse cortical neurones.
J Physiol. 2000 Oct 1;528 Pt 1(Pt 1):39-52. doi: 10.1111/j.1469-7793.2000.00039.x.
3
Protective responses in the ischemic myocardium.
J Clin Invest. 2000 Oct;106(7):813-8. doi: 10.1172/JCI11205.
4
Preconditioning with cortical spreading depression decreases intraischemic cerebral glutamate levels and down-regulates excitatory amino acid transporters EAAT1 and EAAT2 from rat cerebal cortex plasma membranes.
J Neurochem. 2000 Aug;75(2):812-8. doi: 10.1046/j.1471-4159.2000.0750812.x.
5
Enzymatic function of nitric oxide synthases.
Cardiovasc Res. 1999 Aug 15;43(3):521-31. doi: 10.1016/s0008-6363(99)00115-7.
6
Inflammation and stroke: putative role for cytokines, adhesion molecules and iNOS in brain response to ischemia.
Brain Pathol. 2000 Jan;10(1):95-112. doi: 10.1111/j.1750-3639.2000.tb00247.x.
7
Activation of mitogen-activated protein kinases in gerbil hippocampus with ischemic tolerance induced by 3-nitropropionic acid.
Neurosci Lett. 2000 Jan 7;278(1-2):101-4. doi: 10.1016/s0304-3940(99)00906-4.
8
Requirement for nitric oxide activation of p21(ras)/extracellular regulated kinase in neuronal ischemic preconditioning.
Proc Natl Acad Sci U S A. 2000 Jan 4;97(1):436-41. doi: 10.1073/pnas.97.1.436.
9
Respiratory chain inhibition induces tolerance to focal cerebral ischemia.
J Cereb Blood Flow Metab. 1999 Nov;19(11):1229-37. doi: 10.1097/00004647-199911000-00007.
10
MEK1 protein kinase inhibition protects against damage resulting from focal cerebral ischemia.
Proc Natl Acad Sci U S A. 1999 Oct 26;96(22):12866-9. doi: 10.1073/pnas.96.22.12866.
Zotero 插件