Dechambre S D, Duprez T, Grandin C B, Lecouvet F E, Peeters A, Cosnard G
Department of Radiology, Cliniques Universitaires Saint Luc, Brussels, Belgium.
Neuroradiology. 2000 Aug;42(8):608-11. doi: 10.1007/s002340000347.
We describe five cases of high signal in the cerebrospinal fluid (CSF) on fast-FLAIR images 24-48 h after onset of stroke. All the patients had undergone perfusion-weighted MRI within 6 h of the onset of the symptoms. The CSF was far brighter than the cortical gyri. The high signal was diffusely around both cerebral hemispheres in two cases and around one hemisphere in two others; it was focal, around the acute ischaemic lesion, in one. CT was normal in all cases. The CSF high signal was transient, decreasing in extent and intensity with time and resolving completely within 3-6 days. It was not associated with worsening of the clinical state or poor outcome. Our explanation of this phenomena is hypothetical: we speculate that it could be due to disruption of the blood-brain barrier resulting in leakage of protein, gadolinium chelates, or both in to the subarachnoid space. It should not be confused with subarachnoid haemorrhage.
我们描述了5例卒中发病后24 - 48小时快速液体衰减反转恢复（FLAIR）图像上脑脊液（CSF）出现高信号的病例。所有患者在症状发作后6小时内均接受了灌注加权磁共振成像（MRI）检查。脑脊液比脑回亮得多。2例患者双侧大脑半球周围均有弥漫性高信号，另外2例患者仅一侧半球周围有高信号；1例患者的高信号局限于急性缺血性病变周围。所有病例的CT检查均正常。脑脊液高信号是短暂的，其范围和强度随时间逐渐减小，在3 - 6天内完全消失。它与临床状态恶化或预后不良无关。我们对这种现象的解释是推测性的：我们推测这可能是由于血脑屏障破坏导致蛋白质、钆螯合物或两者漏入蛛网膜下腔所致。不应将其与蛛网膜下腔出血相混淆。
