Meena A K, Naidu K S, Murthy J M
Department of Neurology, Nizam's Institute of Medical Sciences, Panjgutta, Hyderabad, 500082, India.
Neurol India. 2000 Sep;48(3):292-4.
Cortical sinovenous thrombosis in a child with nephrotic syndrome and iron deficiency anaemia is described. The most probable mechanism for the hypercoagulable state was thrombocytosis associated with iron deficiency anaemia. The other possible contributing factor might have been the diuretic therapy during the phase of relapse.
本文描述了一名患有肾病综合征和缺铁性贫血的儿童发生皮质静脉窦血栓形成的病例。高凝状态最可能的机制是与缺铁性贫血相关的血小板增多症。另一个可能的促成因素可能是复发期的利尿治疗。
