Hofemeister J, Böhme H
Mol Gen Genet. 1975 Nov 24;141(2):147-61. doi: 10.1007/BF00267680.
Measurements of UV sensitivity of wildtype cells (wt) and UV senistive mutants of E. coli and P. mirabilis suggest that the increased sensitivity of P. mirabilis (wt) is due either to incomplete repair of DNA lesions or to additive lethality probably as a result of UV induction of defective phage(s) present in P. mitabilis (Taubeneck, 1967). Direct estimates of the rate of pyrimidine dimer excision and a comparison of the UV reactivation capacity of E. coli and P. mirabilis for the temperate phages lambda and pi 1, respectively, support this conclusion.
对大肠杆菌和奇异变形杆菌的野生型细胞（wt）以及紫外线敏感突变体的紫外线敏感性测量表明，奇异变形杆菌（wt）敏感性增加的原因要么是DNA损伤修复不完全，要么是可能由于紫外线诱导奇异变形杆菌中存在的缺陷噬菌体而导致的累加致死性（陶贝内克，1967年）。对嘧啶二聚体切除率的直接估计以及分别对大肠杆菌和奇异变形杆菌针对温和噬菌体λ和π1的紫外线复活能力的比较支持了这一结论。
