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本文引用的文献

1
Hostile takeovers: viral appropriation of the NF-kappaB pathway.恶意收购：病毒对核因子κB信号通路的利用
J Clin Invest. 2001 Jan;107(2):143-51. doi: 10.1172/JCI11918.
2
Peroxisome proliferator-activated receptor gamma ligands inhibit development of atherosclerosis in LDL receptor-deficient mice.过氧化物酶体增殖物激活受体γ配体可抑制低密度脂蛋白受体缺陷小鼠动脉粥样硬化的发展。
J Clin Invest. 2000 Aug;106(4):523-31. doi: 10.1172/JCI10370.
3
The NF-kappa B signal transduction pathway in aortic endothelial cells is primed for activation in regions predisposed to atherosclerotic lesion formation.主动脉内皮细胞中的核因子κB信号转导通路在易发生动脉粥样硬化病变形成的区域处于激活准备状态。
Proc Natl Acad Sci U S A. 2000 Aug 1;97(16):9052-7. doi: 10.1073/pnas.97.16.9052.
4
IKKepsilon is part of a novel PMA-inducible IkappaB kinase complex.IKKε是一种新型佛波酯诱导型IκB激酶复合物的一部分。
Mol Cell. 2000 Mar;5(3):513-22. doi: 10.1016/s1097-2765(00)80445-1.
5
Crucial role of the amino-terminal tyrosine residue 42 and the carboxyl-terminal PEST domain of I kappa B alpha in NF-kappa B activation by an oxidative stress.IκBα的氨基末端酪氨酸残基42和羧基末端PEST结构域在氧化应激激活核因子κB中的关键作用。
J Immunol. 2000 Apr 15;164(8):4292-300. doi: 10.4049/jimmunol.164.8.4292.
6
Angiotensin II stimulates endothelial vascular cell adhesion molecule-1 via nuclear factor-kappaB activation induced by intracellular oxidative stress.血管紧张素II通过细胞内氧化应激诱导的核因子-κB激活刺激内皮血管细胞黏附分子-1。
Arterioscler Thromb Vasc Biol. 2000 Mar;20(3):645-51. doi: 10.1161/01.atv.20.3.645.
7
Laminar shear stress inhibits vascular endothelial cell proliferation by inducing cyclin-dependent kinase inhibitor p21(Sdi1/Cip1/Waf1).层流切应力通过诱导细胞周期蛋白依赖性激酶抑制剂p21（Sdi1/Cip1/Waf1）来抑制血管内皮细胞增殖。
Circ Res. 2000 Feb 4;86(2):185-90. doi: 10.1161/01.res.86.2.185.
8
Homocysteine provokes leukocyte-endothelium interaction by downregulation of nitric oxide.同型半胱氨酸通过下调一氧化氮来激发白细胞与内皮细胞的相互作用。
Gen Pharmacol. 1999 Dec;33(6):487-98. doi: 10.1016/s0306-3623(99)00045-2.
9
Cytomegalovirus-mediated modulation of adhesion molecule expression by human arterial and microvascular endothelial cells.巨细胞病毒介导的人动脉和微血管内皮细胞对黏附分子表达的调节
Transplantation. 1999 Dec 15;68(11):1814-8. doi: 10.1097/00007890-199912150-00030.
10
Lysophosphatidic acid activates nuclear factor kappa B and induces proinflammatory gene expression in endothelial cells.溶血磷脂酸激活核因子κB并诱导内皮细胞中促炎基因的表达。
Thromb Haemost. 1999 Nov;82(5):1532-7.

NF-kappaB: pivotal mediator or innocent bystander in atherogenesis?

作者信息

Collins T, Cybulsky M I

机构信息

Department of Pathology, Brigham and Women's Hospital and Harvard Medical School, 221 Longwood Avenue, Boston, Massachusetts 02115, USA.

出版信息

J Clin Invest. 2001 Feb;107(3):255-64. doi: 10.1172/JCI10373.

DOI:10.1172/JCI10373
PMID:11160146
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC199202/
Abstract
摘要