Shapiro K, Caramazza A
Department of Psychology, Harvard University, Cambridge, MA 02138, USA.
Brain Lang. 2001 Feb;76(2):202-12. doi: 10.1006/brln.2000.2397.
Bird, Howard, and Franklin (2000) have proposed a semantic-conceptual explanation of grammatical category-specific deficits that attributes impairments in noun and verb processing to two distinct mechanisms. According to their account, apparent deficits in verb production are not category specific, but rather result from the lower imageability of verbs compared to concrete nouns. Noun deficits are said to result from differences in the distribution of semantic feature types such that damage to sensory features results in disproportionate impairments in naming nouns, especially animate nouns, compared to verbs. However, this hypothesis, which we call the "extended sensory/functional theory" (ESFT), fails on several counts. First, the assumption that representations of living things are more heavily freighted with sensory features than are those of nonliving objects does not have any reliable empirical basis. Second, the ESFT incorrectly predicts associations between deficits in processing sensory features and living things or functional features and nonliving things. Finally, there are numerous cases of patients with grammatical category-specific deficits that do not seem to be consistent with damage at the semantic level. All of this suggests that the ESFT is not a useful model for considering grammatical (or semantic) category-specific deficits.
伯德、霍华德和富兰克林(2000)提出了一种关于语法范畴特异性缺陷的语义概念解释,该解释将名词和动词加工中的损伤归因于两种不同的机制。根据他们的观点,动词产出中明显的缺陷并非范畴特异性的,而是源于与具体名词相比,动词的可想象性较低。名词缺陷据说是由于语义特征类型分布的差异导致的,即与动词相比,感觉特征受损会导致在命名名词(尤其是有生命名词)时出现不成比例的损伤。然而,我们称之为“扩展的感觉/功能理论”(ESFT)的这一假设在几个方面存在问题。首先,认为生物的表征比非生物的表征承载更多感觉特征的假设没有任何可靠的实证依据。其次,ESFT错误地预测了感觉特征加工缺陷与生物之间以及功能特征加工缺陷与非生物之间的关联。最后,有许多语法范畴特异性缺陷的病例似乎与语义层面的损伤不一致。所有这些都表明,ESFT不是一个用于考虑语法(或语义)范畴特异性缺陷的有用模型。