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本文引用的文献
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Molecular analysis of the translational attenuator of a constitutively expressed erm(A) gene from Staphylococcus intermedius.
J Antimicrob Chemother. 2000 Nov;46(5):785-8. doi: 10.1093/jac/46.5.785.
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Prevalence of macrolide-resistance genes in Staphylococcus aureus and Enterococcus faecium isolates from 24 European university hospitals.
J Antimicrob Chemother. 2000 Jun;45(6):891-4. doi: 10.1093/jac/45.6.891.
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Prevalence of resistance to MLS antibiotics in 20 European university hospitals participating in the European SENTRY surveillance programme. Sentry Participants Group.
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Structural alterations in the translational attenuator of constitutively expressed ermC genes.
Antimicrob Agents Chemother. 1999 Jul;43(7):1681-5. doi: 10.1128/AAC.43.7.1681.
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Insights into erythromycin action from studies of its activity as inducer of resistance.
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Naturally occurring Staphylococcus epidermidis plasmid expressing constitutive macrolide-lincosamide-streptogramin B resistance contains a deleted attenuator.
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Nucleotide sequence of ermA, a macrolide-lincosamide-streptogramin B determinant in Staphylococcus aureus.
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Erythromycin-induced stabilization of ermA messenger RNA in Staphylococcus aureus and Bacillus subtilis.
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Bacterial resistance to macrolide, lincosamide, and streptogramin antibiotics by target modification.
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