细菌通过靶点修饰对大环内酯类、林可酰胺类和链阳菌素类抗生素产生耐药性。 - Suppr

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超能文献

Bacterial resistance to macrolide, lincosamide, and streptogramin antibiotics by target modification.

作者信息

Leclercq R, Courvalin P

机构信息

Service de Bactériologie-Virologie-Hygiène, Hôpital Henri Mondor, Université Paris XII, Créteil, France.

出版信息

Antimicrob Agents Chemother. 1991 Jul;35(7):1267-72. doi: 10.1128/AAC.35.7.1267.

DOI:10.1128/AAC.35.7.1267

PMID:1929280

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC245156/

Abstract

摘要

相似文献

Bacterial resistance to macrolide, lincosamide, and streptogramin antibiotics by target modification.细菌通过靶点修饰对大环内酯类、林可酰胺类和链阳菌素类抗生素产生耐药性。

Antimicrob Agents Chemother. 1991 Jul;35(7):1267-72. doi: 10.1128/AAC.35.7.1267.

Intrinsic and unusual resistance to macrolide, lincosamide, and streptogramin antibiotics in bacteria.细菌对大环内酯类、林可酰胺类和链阳菌素类抗生素的内在及异常耐药性。

Antimicrob Agents Chemother. 1991 Jul;35(7):1273-6. doi: 10.1128/AAC.35.7.1273.

Nomenclature for macrolide and macrolide-lincosamide-streptogramin B resistance determinants.大环内酯类及大环内酯-林可酰胺-链阳菌素B耐药决定簇的命名法。

Antimicrob Agents Chemother. 1999 Dec;43(12):2823-30. doi: 10.1128/AAC.43.12.2823.

Inducible resistance to macrolides, lincosamides and streptogramin type B antibiotics: the resistance phenotype, its biological diversity, and structural elements that regulate expression--a review.对大环内酯类、林可酰胺类和B型链阳菌素类抗生素的诱导性耐药：耐药表型、其生物学多样性以及调控表达的结构元件——综述

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Macrolide, lincosamide and streptogramin resistance in Campylobacter jejuni/coli.空肠弯曲菌/结肠弯曲菌对大环内酯类、林可酰胺类和链阳菌素的耐药性

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Resistance to macrolide-lincosamide-streptogramin antibiotics in enterococci from the intestines of animals.动物肠道内肠球菌对大环内酯-林可酰胺-链阳菌素类抗生素的耐药性

Res Vet Sci. 1982 Jul;33(1):70-2.

[Resistance to macrolides].[对大环内酯类药物的耐药性]

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New macrolides active against Streptococcus pyogenes with inducible or constitutive type of macrolide-lincosamide-streptogramin B resistance.对具有诱导型或组成型大环内酯-林可酰胺-链阳菌素B耐药性的化脓性链球菌有效的新型大环内酯类药物。

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[Inducible resistance to macrolide-lincosamide-streptogramin type B antibiotics in Bacillus licheniformis: common structures of macrolide antibiotics capable of inducing the resistance].地衣芽孢杆菌对大环内酯-林可酰胺-链阳菌素B型抗生素的诱导抗性：能够诱导该抗性的大环内酯类抗生素的共同结构

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Prevalence and characterization of the mechanisms of macrolide, lincosamide, and streptogramin resistance in isolates of Streptococcus pneumoniae.肺炎链球菌分离株中大环内酯类、林可酰胺类及链阳菌素耐药机制的发生率与特征

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本文引用的文献

ELIMINATION OF DRUG RESISTANCE OF STAPHYLOCOCCUS AUREUS BY TREATMENT WITH ACRIFLAVINE.吖啶黄治疗消除金黄色葡萄球菌的耐药性

J Bacteriol. 1963 Jul;86(1):162-4. doi: 10.1128/jb.86.1.162-164.1963.

The erythromycin group of antibiotics.红霉素类抗生素。

Br Med J. 1957 Jul 13;2(5036):57-63. doi: 10.1136/bmj.2.5036.57.

Development of resistance and cross-resistance in vitro to erythromycin, carbomycin, spiramycin, oleandomycin and streptogramin.体外对红霉素、碳霉素、螺旋霉素、竹桃霉素和链阳霉素的耐药性及交叉耐药性的发展

Proc Soc Exp Biol Med. 1956 Nov;93(2):388-93. doi: 10.3181/00379727-93-22766.

[In vitro antagonism between erythromycin and spiramycin].[红霉素与螺旋霉素的体外拮抗作用]

Ann Inst Pasteur (Paris). 1956 Jun;90(6):787-90.

Classification of macrolide-lincosamide-streptogramin-B-type antibiotic resistance determinants.大环内酯-林可酰胺-链阳菌素B型抗生素耐药决定因子的分类

Ann Microbiol (Paris). 1981 Nov-Dec;132 B(3):441-54.

Characterization of pBFTM10, a clindamycin-erythromycin resistance transfer factor from Bacteroides fragilis.脆弱拟杆菌中克林霉素-红霉素耐药转移因子pBFTM10的特性分析

J Bacteriol. 1982 Aug;151(2):686-91. doi: 10.1128/jb.151.2.686-691.1982.

Conjugative transfer of multiple antibiotic resistance markers in Streptococcus pneumoniae.肺炎链球菌中多种抗生素抗性标记的接合转移

J Bacteriol. 1980 Jul;143(1):313-20. doi: 10.1128/jb.143.1.313-320.1980.

Posttranscriptional modification of mRNA conformation: mechanism that regulates erythromycin-induced resistance.mRNA构象的转录后修饰：调节红霉素诱导抗性的机制

Proc Natl Acad Sci U S A. 1980 Dec;77(12):7079-83. doi: 10.1073/pnas.77.12.7079.

Translational attenuation of ermC: a deletion analysis.ermC的翻译弱化：缺失分析

Mol Gen Genet. 1982;186(2):204-16. doi: 10.1007/BF00331851.

Large transmissible clindamycin resistance plasmid in Bacteroides ovatus.卵形拟杆菌中的大型可传播克林霉素耐药质粒。

J Bacteriol. 1984 May;158(2):739-41. doi: 10.1128/jb.158.2.739-741.1984.

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