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Enzyme release during myocardial anoxia: a study of metabolic protection.

作者信息

Hearse D J, Humphrey S M

出版信息

J Mol Cell Cardiol. 1975 Jul;7(7):463-82. doi: 10.1016/0022-2828(75)90164-9.

DOI:10.1016/0022-2828(75)90164-9
PMID:1152074
Abstract
摘要

相似文献

1
Enzyme release during myocardial anoxia: a study of metabolic protection.
J Mol Cell Cardiol. 1975 Jul;7(7):463-82. doi: 10.1016/0022-2828(75)90164-9.
2
Abrupt reoxygenation of the anoxic potassium-arrested perfused rat heart: a study of myocardial enzyme release.
J Mol Cell Cardiol. 1973 Aug;5(4):395-407. doi: 10.1016/0022-2828(73)90030-8.
3
Myocardial enzyme loss: factors leading to reduction or exacerbation of release.心肌酶流失：导致释放减少或加剧的因素。
Recent Adv Stud Cardiac Struct Metab. 1975;7:327-34.
4
Sensitivity of enzymatic release due to ischemia in the isolated rat heart.离体大鼠心脏中缺血所致酶释放的敏感性。
Acta Physiol Lat Am. 1982;32(4):303-11.
5
[A method for the gradual determination of hypoxic damage in the Langendorff isolated guinea pig heart].
Arzneimittelforschung. 1973 Jul;23(7):926-9.
6
[The enzymatic diagnosis of myocardial infarction. I. The activity of glutamic oxaloacetic transaminase, of lactate dehydrogenase, of alpha-hydroxybutyrate dehydrogenase and creatine phosphokinase in myocardial infarction].[心肌梗死的酶学诊断。I. 心肌梗死时谷氨酸草酰乙酸转氨酶、乳酸脱氢酶、α-羟丁酸脱氢酶及肌酸磷酸激酶的活性]
Enzymol Biol Clin (Basel). 1966;6(1):25-35.
7
Serum creatine phosphokinase estimations in myocardial infarction.心肌梗死时血清肌酸磷酸激酶的测定
Br Heart J. 1965 Sep;27(5):740-7. doi: 10.1136/hrt.27.5.740.
8
Experimental right-ventricular infarction in rats by abdominodiaphragmal access to the heart. A new technique in the study of infarcted myocardium.通过腹膈途径对大鼠进行实验性右心室梗死。一种研究梗死心肌的新技术。
Res Exp Med (Berl). 1983;182(2):105-9. doi: 10.1007/BF01851116.
9
Effect of a thrombolytic agent (urokinase) on necrosis after acute myocardial infarction.溶栓剂（尿激酶）对急性心肌梗死后坏死的影响。
Recent Adv Stud Cardiac Struct Metab. 1975;10:319-29.
10
[The enzymatic diagnosis of myocardial infarction. IV. Serum enzyme pattern in myocardial infarction].[心肌梗死的酶学诊断。IV. 心肌梗死时的血清酶谱]
Enzymol Biol Clin (Basel). 1966;6(1):66-78.

引用本文的文献

1
A reproducible method for biochemical, histological and functional assessment of the effects of ischaemia-reperfusion syndrome in the lower limbs.一种可重现的方法，用于评估下肢缺血再灌注综合征对生化、组织学和功能的影响。
Sci Rep. 2021 Sep 29;11(1):19325. doi: 10.1038/s41598-021-98887-9.
2
Fifty percent nitrous oxide depresses recovery from anoxic heart failure induced by 100% nitrous oxide.
J Anesth. 1987 Sep 1;1(2):119-24. doi: 10.1007/s0054070010119.
3
Glucose and glycogen utilisation in myocardial ischemia--changes in metabolism and consequences for the myocyte.心肌缺血时葡萄糖和糖原的利用——代谢变化及对心肌细胞的影响
Mol Cell Biochem. 1998 Mar;180(1-2):3-26.
4
Reduction of cell injury in hypoxic cultures of rat myocardial cells by methylprednisolone.甲基强的松龙减轻大鼠心肌细胞缺氧培养中的细胞损伤。
In Vitro. 1980 Feb;16(2):93-6. doi: 10.1007/BF02831498.
5
Ineffectiveness of methylprednisolone to reduce infarct size in experimental coronary occlusion.甲基强的松龙在实验性冠状动脉闭塞中减少梗死面积的无效性。
Basic Res Cardiol. 1982 Mar-Apr;77(2):182-7. doi: 10.1007/BF01908171.
6
Magnesium and the calcium paradox: the occurrence of "spasmodic contractions" during Ca2+-Mg2+ -free perfusion of isolated rat heart.镁与钙悖论：离体大鼠心脏在无钙镁灌注期间出现“痉挛性收缩”
Basic Res Cardiol. 1983 Mar-Apr;78(2):227-38. doi: 10.1007/BF01906675.
7
Relation of enzyme release and ATP content in digoxin-intoxicated guinea-pig hearts.地高辛中毒豚鼠心脏中酶释放与ATP含量的关系
Basic Res Cardiol. 1982 Jul-Aug;77(4):449-59. doi: 10.1007/BF02005344.
8
Studies on myocardial protection by cold fluorocarbon (made in China) cardioplegic solution: experiment on isolated rabbit heart.国产冷氟碳停搏液对心肌保护作用的研究：家兔离体心脏实验
Acta Acad Med Wuhan. 1985;5(2):71-7. doi: 10.1007/BF02888631.
9
Glucocorticoids in myocardial and cerebral infarction.心肌梗死和脑梗死中的糖皮质激素
Agents Actions. 1986 Jan;17(3-4):278-83. doi: 10.1007/BF01982620.
10
Beneficial effect of amosulalol and phentolamine on post-hypoxic recovery of contractile force and energy metabolism in rabbit hearts.阿唑洛尔和酚妥拉明对兔心脏缺氧后收缩力和能量代谢恢复的有益作用。
Br J Pharmacol. 1989 Jun;97(2):513-23. doi: 10.1111/j.1476-5381.1989.tb11980.x.