Rutherford A W, Krieger-Liszkay A
Section de Bioénergétique, DBCM, CEA CNRS URA 2096, CE Saclay, 91191, Gif-sur-Yvette, France.
Trends Biochem Sci. 2001 Nov;26(11):648-53. doi: 10.1016/s0968-0004(01)01953-3.
Some herbicides act by binding to the exchangeable quinone site in the photosystem II (PSII) reaction centre, thus blocking electron transfer. In this article, it is hypothesized that the plant is killed by light-induced oxidative stress initiated by damage caused by formation of singlet oxygen in the reaction centre itself. This occurs when light-induced charge pairs in herbicide-inhibited PSII decay by a charge recombination route involving the formation of a chlorophyll triplet state that is able to activate oxygen. The binding of phenolic herbicides favours this pathway, thus increasing the efficiency of photodamage in this class of herbicides.
一些除草剂通过与光系统II（PSII）反应中心的可交换醌位点结合来发挥作用，从而阻断电子传递。在本文中，有人提出假设，植物是被反应中心自身形成单线态氧所造成的损伤引发的光诱导氧化应激杀死的。当除草剂抑制的PSII中光诱导电荷对通过涉及形成能够激活氧的叶绿素三重态的电荷复合途径衰减时，就会发生这种情况。酚类除草剂的结合有利于这一途径，从而提高了这类除草剂的光损伤效率。
