波特综合征。羊水过少引起的非肾脏特征。
Potter syndrome. Nonrenal features induced by oligoamnios.
作者信息
Fantel A G, Shepard T H
出版信息
Am J Dis Child. 1975 Nov;129(11):1346-7. doi: 10.1001/archpedi.1975.02120480060015.
Abstract
A fetus with normal kidneys and the nonrenal features of Potter syndrome is presented. The case is made that these features resulted from uterine compression caused by premature rupture of the gestational membranes and subsequent leakage of amniotic fluid.
摘要
本文介绍了一名具有正常肾脏但伴有波特综合征非肾脏特征的胎儿。据推测，这些特征是由胎膜早破导致的子宫受压以及随后的羊水渗漏引起的。
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