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Discrepancy between antithrombin activity methods revealed in Antithrombin Stockholm: do factor Xa-based methods overestimate antithrombin activity in some patients?

作者信息

Ungerstedt Johanna S, Schulman Sam, Egberg Nils, Antovic Jovan, Blombäck Nargareta

出版信息

Blood. 2002 Mar 15;99(6):2271-2. doi: 10.1182/blood-2001-11-0047.

DOI:10.1182/blood-2001-11-0047
PMID:11902137
Abstract
摘要

相似文献

1
Discrepancy between antithrombin activity methods revealed in Antithrombin Stockholm: do factor Xa-based methods overestimate antithrombin activity in some patients?《抗凝血酶斯德哥尔摩研究》揭示的抗凝血酶活性检测方法间的差异：基于因子Xa的检测方法是否在某些患者中高估了抗凝血酶活性？
Blood. 2002 Mar 15;99(6):2271-2. doi: 10.1182/blood-2001-11-0047.
2
Detecting antithrombin deficiency may be a difficult task--more than one test is necessary.检测抗凝血酶缺乏症可能是一项艰巨的任务——需要不止一项检测。
J Thromb Haemost. 2007 Mar;5(3):617-8. doi: 10.1111/j.1538-7836.2007.02395.x. Epub 2007 Jan 10.
3
The progressive antithrombin (antithrombin-III, heparin-cofactor).
Haematologia (Budap). 1974;8(1-4):455-9.
4
Abnormal antithrombin III (antithrombin III "Budapest") as a cause of a familial thrombophilia.异常抗凝血酶III（抗凝血酶III“布达佩斯型”）作为家族性血栓形成倾向的一个病因
Thromb Diath Haemorrh. 1974 Sep 30;32(1):105-15.
5
Prevalence of antithrombin deficiency in the healthy population.
Br J Haematol. 1994 May;87(1):106-12. doi: 10.1111/j.1365-2141.1994.tb04878.x.
6
An antithrombin III assay based on factor Xa inhibition provides a more reliable test to identify congenital antithrombin III deficiency than an assay based on thrombin inhibition.基于因子Xa抑制的抗凝血酶III检测比基于凝血酶抑制的检测能更可靠地识别先天性抗凝血酶III缺乏。
Thromb Haemost. 1993 Mar 1;69(3):231-5.
7
THROMBOPHILIA CAUSED BY INHERITABLE DEFICIENCY OF BLOOD ANTITHROMBIN.遗传性抗凝血酶缺乏引起的血栓形成倾向
Scand J Clin Lab Invest. 1965;17:92. doi: 10.3109/00365516509077290.
8
Molecular basis of antithrombin deficiency in four Japanese patients with antithrombin gene abnormalities including two novel mutations.4例抗凝血酶基因异常的日本患者抗凝血酶缺乏的分子基础，包括2种新突变。
Am J Hematol. 2007 Aug;82(8):702-5. doi: 10.1002/ajh.20924.
9
Characterization of two novel mutations of the antithrombin gene observed in Japanese thrombophilic patients.
Thromb Res. 2005;115(5):351-8. doi: 10.1016/j.thromres.2004.08.025.
10
The antithrombin P1 residue is important for target proteinase specificity but not for heparin activation of the serpin. Characterization of P1 antithrombin variants with altered proteinase specificity but normal heparin activation.抗凝血酶的P1残基对靶蛋白酶特异性很重要，但对丝氨酸蛋白酶抑制剂的肝素激活不重要。具有改变的蛋白酶特异性但肝素激活正常的P1抗凝血酶变体的表征。
Biochemistry. 2001 Jun 5;40(22):6670-9. doi: 10.1021/bi002933d.

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Functional Characterization of Antithrombin Mutations by Monitoring of Thrombin Inhibition Kinetics.通过监测凝血酶抑制动力学对抗凝血酶突变进行功能表征。
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