Paintal I S, Minina R J, Ramchandani I K
Indian J Physiol Pharmacol. 1975 Oct-Dec;19(4):199-202.
Platelet counts of 42 alcoholics studied showed that 88% of them had thrombocytopenia. This hematological response to ethanol was dose related. Liver function assessed by biochemical tests showed no liver parenchymal damage and hence could not explain the thrombocytopenia. It has been suggested that this effect may result from a direct effect of ethanol on bone marrow from shortened platelet survival or their sequestration.
对42名酗酒者的血小板计数研究表明，其中88%的人患有血小板减少症。这种对乙醇的血液学反应与剂量相关。通过生化测试评估的肝功能显示没有肝实质损伤，因此无法解释血小板减少症。有人认为，这种效应可能是由于乙醇对骨髓的直接作用、血小板存活时间缩短或血小板被隔离所致。
