Jaks, STATs, Cytokines, and Sepsis.
作者信息
Scott Melanie J, Godshall Christopher J, Cheadle William G
机构信息
Department of Surgery and Price Institute for Surgical Research, University of Louisville School of Medicine, Louisville, Kentucky 40292, USA.
出版信息
Clin Diagn Lab Immunol. 2002 Nov;9(6):1153-9. doi: 10.1128/cdli.9.6.1153-1159.2002.
Abstract
摘要
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本文引用的文献
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Six at six: interleukin-6 measured 6 h after the initiation of sepsis predicts mortality over 3 days.六小时的六指标：脓毒症开始后6小时测量的白细胞介素-6可预测3天内的死亡率。
Shock. 2002 Jun;17(6):463-7. doi: 10.1097/00024382-200206000-00004.
2
Involvement of suppressor of cytokine signaling-3 as a mediator of the inhibitory effects of IL-10 on lipopolysaccharide-induced macrophage activation.细胞因子信号转导抑制因子3作为白细胞介素-10对脂多糖诱导的巨噬细胞激活抑制作用的介质参与其中。
J Immunol. 2002 Jun 15;168(12):6404-11. doi: 10.4049/jimmunol.168.12.6404.
3
Reduced expression of STAT4 and IFN-gamma in macrophages from BALB/c mice.BALB/c小鼠巨噬细胞中STAT4和干扰素-γ的表达降低。
J Immunol. 2002 Jun 1;168(11):5477-82. doi: 10.4049/jimmunol.168.11.5477.
4
Impaired monocyte IL-12 production before surgery as a predictive factor for the lethal outcome of postoperative sepsis.术前单核细胞白细胞介素-12产生受损作为术后脓毒症致死结局的预测因素。
Ann Surg. 2002 Apr;235(4):560-7. doi: 10.1097/00000658-200204000-00015.
5
Interleukin-10: A complex role in the pathogenesis of sepsis syndromes and its potential as an anti-inflammatory drug.白细胞介素-10：在脓毒症综合征发病机制中的复杂作用及其作为抗炎药物的潜力。
Crit Care Med. 2002 Jan;30(1 Supp):S58-S63.
6
Suppressors of cytokine signaling (SOCS): inhibitors of the JAK/STAT pathway.细胞因子信号转导抑制因子（SOCS）：JAK/STAT信号通路的抑制剂
Shock. 2002 Feb;17(2):83-90. doi: 10.1097/00024382-200202000-00001.
7
Role of IFN-gamma in bacterial containment in a model of intra-abdominal sepsis.γ干扰素在腹腔内脓毒症模型中细菌控制方面的作用
Shock. 2001 Dec;16(6):425-9. doi: 10.1097/00024382-200116060-00004.
8
Severe sepsis induces deficient interferon-gamma and interleukin-12 production, but interleukin-12 therapy improves survival in peritonitis.严重脓毒症会导致γ-干扰素和白细胞介素-12生成不足，但白细胞介素-12治疗可提高腹膜炎患者的生存率。
Am J Surg. 2001 Nov;182(5):491-7. doi: 10.1016/s0002-9610(01)00754-1.
9
STAT4 is required for antibacterial defense but enhances mortality during polymicrobial sepsis.信号转导和转录激活因子4（STAT4）是抗菌防御所必需的，但会增加多重微生物败血症期间的死亡率。
Clin Diagn Lab Immunol. 2001 Nov;8(6):1044-8. doi: 10.1128/CDLI.8.6.1044-1048.2001.
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Regulation of interleukin (IL)-18 receptor alpha chain expression on CD4(+) T cells during T helper (Th)1/Th2 differentiation. Critical downregulatory role of IL-4.辅助性T细胞（Th）1/Th2分化过程中白细胞介素（IL）-18受体α链在CD4(+) T细胞上的表达调控。IL-4的关键下调作用。
J Exp Med. 2001 Jul 16;194(2):143-53. doi: 10.1084/jem.194.2.143.
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