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2
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Infect Immun. 1978 Mar;19(3):763-70. doi: 10.1128/iai.19.3.763-770.1978.
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本文引用的文献

1
Neutrophil-Kupffer cell interaction: a critical component of host defenses to systemic bacterial infections.中性粒细胞-库普弗细胞相互作用：宿主抵御全身性细菌感染的关键组成部分。
J Leukoc Biol. 2002 Aug;72(2):239-48.
2
Phenotype of mice and macrophages deficient in both phagocyte oxidase and inducible nitric oxide synthase.吞噬细胞氧化酶和诱导型一氧化氮合酶均缺乏的小鼠和巨噬细胞的表型。
Immunity. 1999 Jan;10(1):29-38. doi: 10.1016/s1074-7613(00)80004-7.
3
Regulation and functional involvement of macrophage scavenger receptor MARCO in clearance of bacteria in vivo.巨噬细胞清道夫受体MARCO在体内细菌清除中的调节作用及功能参与
J Immunol. 1999 Jan 15;162(2):939-47.
4
Murine listeriosis as a model of antimicrobial defense.鼠类李斯特菌病作为抗菌防御的模型
Immunol Rev. 1997 Aug;158:27-36. doi: 10.1111/j.1600-065x.1997.tb00989.x.
5
Mice lacking reduced nicotinamide adenine dinucleotide phosphate oxidase activity show increased susceptibility to early infection with Listeria monocytogenes.缺乏还原型烟酰胺腺嘌呤二核苷酸磷酸氧化酶活性的小鼠对单核细胞增生李斯特菌早期感染的易感性增加。
J Immunol. 1997 Jun 15;158(12):5581-3.
6
Inter-relationship among macrophages, natural killer cells and neutrophils in early stages of Listeria resistance.李斯特菌抗性早期阶段巨噬细胞、自然杀伤细胞和中性粒细胞之间的相互关系。
Curr Opin Immunol. 1997 Feb;9(1):35-43. doi: 10.1016/s0952-7915(97)80156-2.
7
Neutrophils are essential for early anti-Listeria defense in the liver, but not in the spleen or peritoneal cavity, as revealed by a granulocyte-depleting monoclonal antibody.一种粒细胞清除单克隆抗体显示，中性粒细胞对于肝脏早期抗李斯特菌防御至关重要，但对脾脏或腹腔的防御并非如此。
J Exp Med. 1994 Jan 1;179(1):259-68. doi: 10.1084/jem.179.1.259.
8
Neutrophils are involved in acute, nonspecific resistance to Listeria monocytogenes in mice.中性粒细胞参与小鼠对单核细胞增生李斯特菌的急性非特异性抵抗。
Infect Immun. 1993 Dec;61(12):5090-6. doi: 10.1128/iai.61.12.5090-5096.1993.
9
Administration of anti-granulocyte mAb RB6-8C5 impairs the resistance of mice to Listeria monocytogenes infection.给予抗粒细胞单克隆抗体RB6-8C5会损害小鼠对单核细胞增生李斯特菌感染的抵抗力。
J Immunol. 1994 Feb 15;152(4):1836-46.
10
Roles of Listeria monocytogenes virulence factors in survival: virulence factors distinct from listeriolysin are needed for the organism to survive an early neutrophil-mediated host defense mechanism.单核细胞增生李斯特菌毒力因子在生存中的作用：该生物体要在早期中性粒细胞介导的宿主防御机制中存活，需要不同于溶血素的毒力因子。
Infect Immun. 1992 Mar;60(3):951-7. doi: 10.1128/iai.60.3.951-957.1992.

小鼠中烟酰胺腺嘌呤二核苷酸磷酸（NADPH）氧化酶依赖性抗李斯特菌病的表达在肝脏感染的最初6至12小时内出现。

Expression of NADPH oxidase-dependent resistance to listeriosis in mice occurs during the first 6 to 12 hours of liver infection.

作者信息

LaCourse Ronald, Ryan Lynn, North Robert J

机构信息

The Trudeau Institute, Saranac Lake, New York 12983, USA.

出版信息

Infect Immun. 2002 Dec;70(12):7179-81. doi: 10.1128/IAI.70.12.7179-7181.2002.

DOI:10.1128/IAI.70.12.7179-7181.2002
PMID:12438407
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC133076/
Abstract

Wild-type mice inoculated with Listeria monocytogenes intravenously were capable of reducing the bacterial load in their livers by 90% within 6 h. In contrast, mice with deletions of the gene for NADPH oxidase were incapable of expressing this early oxygen-dependent anti-Listeria defense and consequently showed higher levels of liver infection at later times.

摘要

静脉注射单核细胞增生李斯特菌的野生型小鼠能够在6小时内将肝脏中的细菌载量降低90%。相比之下，缺失NADPH氧化酶基因的小鼠无法表达这种早期的氧依赖性抗李斯特菌防御机制，因此在后期显示出更高水平的肝脏感染。