Popova N K, Nikulina E M, Arav V A, Kudriavtseva N N
Fiziol Zh SSSR Im I M Sechenova. 1975 Feb;61(2):183-6.
Electrolytic lesion of the midbrain raphe nuclei in rats, lowering serotonin level in the forebrain, elicited mouse-killing behavior. After the lesion, 50% of previously non-killers rats became mouse-killers. 5-Hydroxytryptophan (100 mg/kg) administration elevated serotonin level to normal values and completely blocked predatory aggression. p-Chlorophenylalanine produced obvious reduction in brain serotonin and slightly stimulated aggressive behavior. These results suggest that the brain serotonin participates as an inhibitory factor in mouse-killing behavior in rats.
对大鼠中脑缝际核进行电解损伤，降低前脑中的血清素水平，引发了杀鼠行为。损伤后，50%之前不杀鼠的大鼠变成了杀鼠者。给予5-羟色氨酸（100毫克/千克）可使血清素水平升高至正常值，并完全阻断捕食性攻击行为。对氯苯丙氨酸使脑中血清素明显减少，并轻微刺激攻击行为。这些结果表明，脑血清素作为一种抑制因子参与大鼠的杀鼠行为。
