Ellickson P L, Hays R D
Social Policy Department, RAND, Santa Monica, CA 90407-2138.
Health Psychol. 1992;11(6):377-85. doi: 10.1037//0278-6133.11.6.377.
We tested a model of the process of becoming involved with drugs during junior high. The sample included 698 students who were not using alcohol, cigarettes, or marijuana during Grade 7 (T1); the follow-up data points were 12 and 15 months later (T2 and T3). The final model, which predicted 72% of the variance in drug use at T3, provides support for hypotheses drawn from both social and cognitive theories. Weak familial and school attachments fostered use by increasing the likelihood of exposure to pro-drug social influences (drug use offers); weak bonds with school also directly affected cognitive motivations (lower resistance self-efficacy [RSE] and more positive outcome expectancies). In turn, social influences at T1 played a dominant role in initial use at T2, but cognitive motivations were also significant. At T3, prior use assumed the most prominent position. Drug-specific measures of RSE and expected use directly affected later use of that substance. The results indicate that both generic and drug-specific effects are needed to explain adolescent drug use. Implications for prevention programs are discussed.
我们测试了一个初中阶段涉毒过程的模型。样本包括698名在七年级(T1)时未使用酒精、香烟或大麻的学生;随访数据点分别在12个月和15个月后(T2和T3)。最终模型预测了T3时72%的药物使用差异,为来自社会和认知理论的假设提供了支持。薄弱的家庭和学校依恋关系通过增加接触亲毒品社会影响(毒品使用机会)的可能性来助长毒品使用;与学校的薄弱联系也直接影响认知动机(较低的抵抗自我效能感[RSE]和更积极的结果期望)。反过来,T1时的社会影响在T2时的初次使用中起主导作用,但认知动机也很重要。在T3时,先前的使用占据了最突出的位置。RSE的药物特异性测量和预期使用直接影响该物质的后期使用。结果表明,需要综合一般影响和药物特异性影响来解释青少年的药物使用情况。文中还讨论了对预防项目的启示。