Reovirus receptors and pathogenesis.
作者信息
Forrest J Craig, Dermody Terence S
机构信息
Department of Microbiology and Immunology, Vanderbilt University School of Medicine, Nashville, Tennessee 37232, USA.
出版信息
J Virol. 2003 Sep;77(17):9109-15. doi: 10.1128/jvi.77.17.9109-9115.2003.
Abstract
摘要
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Reovirus receptors and pathogenesis.呼肠孤病毒受体与发病机制。
J Virol. 2003 Sep;77(17):9109-15. doi: 10.1128/jvi.77.17.9109-9115.2003.
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本文引用的文献
1
Utilization of sialic acid as a coreceptor is required for reovirus-induced biliary disease.呼肠孤病毒诱导的胆道疾病需要利用唾液酸作为共受体。
J Clin Invest. 2003 Jun;111(12):1823-33. doi: 10.1172/JCI16303.
2
Viral interactions with receptors in cell junctions and effects on junctional stability.病毒与细胞连接中的受体相互作用及其对连接稳定性的影响。
Dev Cell. 2002 Oct;3(4):462-4. doi: 10.1016/s1534-5807(02)00298-8.
3
Reovirus-induced apoptosis requires mitochondrial release of Smac/DIABLO and involves reduction of cellular inhibitor of apoptosis protein levels.呼肠孤病毒诱导的细胞凋亡需要线粒体释放Smac/DIABLO，并涉及细胞凋亡抑制蛋白水平的降低。
J Virol. 2002 Nov;76(22):11414-24. doi: 10.1128/jvi.76.22.11414-11424.2002.
4
Interferon regulatory factor-1, interferon-beta, and reovirus-induced myocarditis.干扰素调节因子-1、β-干扰素与呼肠孤病毒诱导的心肌炎
Virology. 2002 Jun 20;298(1):20-9. doi: 10.1006/viro.2002.1470.
5
Disruption of adherens junctions liberates nectin-1 to serve as receptor for herpes simplex virus and pseudorabies virus entry.黏附连接的破坏会释放nectin-1，使其作为单纯疱疹病毒和伪狂犬病病毒进入细胞的受体。
J Virol. 2002 Jul;76(14):7203-8. doi: 10.1128/jvi.76.14.7203-7208.2002.
6
Structure of the reovirus membrane-penetration protein, Mu1, in a complex with is protector protein, Sigma3.呼肠孤病毒膜穿透蛋白Mu1与保护蛋白Sigma3形成的复合物的结构。
Cell. 2002 Jan 25;108(2):283-95. doi: 10.1016/s0092-8674(02)00612-8.
7
Virion disassembly is required for apoptosis induced by reovirus.呼肠孤病毒诱导的细胞凋亡需要病毒粒子的解体。
J Virol. 2002 Feb;76(4):1632-41. doi: 10.1128/jvi.76.4.1632-1641.2002.
8
Crystal structure of reovirus attachment protein sigma1 reveals evolutionary relationship to adenovirus fiber.呼肠孤病毒附着蛋白σ1的晶体结构揭示了其与腺病毒纤维的进化关系。
EMBO J. 2002 Jan 15;21(1-2):1-11. doi: 10.1093/emboj/21.1.1.
9
The coxsackievirus and adenovirus receptor is a transmembrane component of the tight junction.柯萨奇病毒和腺病毒受体是紧密连接的一种跨膜成分。
Proc Natl Acad Sci U S A. 2001 Dec 18;98(26):15191-6. doi: 10.1073/pnas.261452898. Epub 2001 Dec 4.
10
Reovirus infection activates JNK and the JNK-dependent transcription factor c-Jun.呼肠孤病毒感染会激活JNK以及依赖JNK的转录因子c-Jun。
J Virol. 2001 Dec;75(23):11275-83. doi: 10.1128/JVI.75.23.11275-11283.2001.
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