Oxygen toxicity and the health and survival of eukaryote cells: a new piece is added to the puzzle.
作者信息
Archibald F
机构信息
Department of Natural Resource Sciences, McGill University, Quebec, QC, Canada H9X 3V9.
出版信息
Proc Natl Acad Sci U S A. 2003 Sep 2;100(18):10141-3. doi: 10.1073/pnas.1934513100. Epub 2003 Aug 25.
Abstract
摘要
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Oxygen toxicity and the health and survival of eukaryote cells: a new piece is added to the puzzle.氧中毒与真核细胞的健康和存活：拼图增添新板块。
Proc Natl Acad Sci U S A. 2003 Sep 2;100(18):10141-3. doi: 10.1073/pnas.1934513100. Epub 2003 Aug 25.
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Suppression of oxidative damage by Saccharomyces cerevisiae ATX2, which encodes a manganese-trafficking protein that localizes to Golgi-like vesicles.酿酒酵母ATX2对氧化损伤的抑制作用，该基因编码一种定位于高尔基体样囊泡的锰转运蛋白。
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Manganese superoxide dismutase in Saccharomyces cerevisiae acquires its metal co-factor through a pathway involving the Nramp metal transporter, Smf2p.酿酒酵母中的锰超氧化物歧化酶通过一条涉及Nramp金属转运蛋白Smf2p的途径获取其金属辅因子。
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Regulation of manganese superoxide dismutase in Saccharomyces cerevisiae. The role of respiratory chain activity.酿酒酵母中锰超氧化物歧化酶的调控。呼吸链活性的作用。
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Manganese activation of superoxide dismutase 2 in Saccharomyces cerevisiae requires MTM1, a member of the mitochondrial carrier family.酿酒酵母中超氧化物歧化酶2的锰激活需要线粒体载体家族成员MTM1。
Proc Natl Acad Sci U S A. 2003 Sep 2;100(18):10353-7. doi: 10.1073/pnas.1632471100. Epub 2003 Jul 30.
2
Manganese superoxide dismutase-mediated gene expression in radiation-induced adaptive responses.锰超氧化物歧化酶介导的辐射诱导适应性反应中的基因表达。
Mol Cell Biol. 2003 Apr;23(7):2362-78. doi: 10.1128/MCB.23.7.2362-2378.2003.
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Increased manganese superoxide dismutase (SOD-2) is part of the mechanism for prostate tumor suppression by Mac25/insulin-like growth factor binding-protein-related protein-1.锰超氧化物歧化酶（SOD-2）水平升高是Mac25/胰岛素样生长因子结合蛋白相关蛋白-1抑制前列腺肿瘤机制的一部分。
Oncogene. 2003 Feb 20;22(7):1024-34. doi: 10.1038/sj.onc.1206210.
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Manganese superoxide dismutase in Saccharomyces cerevisiae acquires its metal co-factor through a pathway involving the Nramp metal transporter, Smf2p.酿酒酵母中的锰超氧化物歧化酶通过一条涉及Nramp金属转运蛋白Smf2p的途径获取其金属辅因子。
J Biol Chem. 2001 Dec 14;276(50):47556-62. doi: 10.1074/jbc.M108923200. Epub 2001 Oct 15.
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Knockout mice heterozygous for Sod2 show alterations in cardiac mitochondrial function and apoptosis.Sod2基因杂合敲除小鼠表现出心脏线粒体功能和细胞凋亡的改变。
Am J Physiol Heart Circ Physiol. 2001 Sep;281(3):H1422-32. doi: 10.1152/ajpheart.2001.281.3.H1422.
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A fraction of yeast Cu,Zn-superoxide dismutase and its metallochaperone, CCS, localize to the intermembrane space of mitochondria. A physiological role for SOD1 in guarding against mitochondrial oxidative damage.一部分酵母铜锌超氧化物歧化酶及其金属伴侣蛋白CCS定位于线粒体的膜间隙。超氧化物歧化酶1在预防线粒体氧化损伤方面的生理作用。
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Increased mitochondrial oxidative stress in the Sod2 (+/-) mouse results in the age-related decline of mitochondrial function culminating in increased apoptosis.Sod2基因杂合子小鼠线粒体氧化应激增加，导致与年龄相关的线粒体功能衰退，最终凋亡增加。
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Mice with a partial deficiency of manganese superoxide dismutase show increased vulnerability to the mitochondrial toxins malonate, 3-nitropropionic acid, and MPTP.锰超氧化物歧化酶部分缺乏的小鼠对线粒体毒素丙二酸、3-硝基丙酸和MPTP的易感性增加。
Exp Neurol. 2001 Jan;167(1):189-95. doi: 10.1006/exnr.2000.7525.
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Effect of stress on the life span of the yeast Saccharomyces cerevisiae.应激对酿酒酵母寿命的影响。
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