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本文引用的文献
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Brief, repeated, oxygen-glucose deprivation episodes protect neurotransmission from a longer ischemic episode in the in vitro hippocampus: role of adenosine receptors.
Br J Pharmacol. 2003 Sep;140(2):305-14. doi: 10.1038/sj.bjp.0705442. Epub 2003 Aug 11.
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The selective A2A receptor antagonist SCH 58261 reduces striatal transmitter outflow, turning behavior and ischemic brain damage induced by permanent focal ischemia in the rat.
Brain Res. 2003 Jan 10;959(2):243-50. doi: 10.1016/s0006-8993(02)03753-8.
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Suppression of presynaptic responses to adenosine by activation of NMDA receptors.
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Long-term potentiation protects rat hippocampal slices from the effects of acute hypoxia.
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NMDA-induced changes in a cortical network in vivo are prevented by AMPA.
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NMDA receptor antagonism, but not AMPA receptor antagonism attenuates induced ischaemic tolerance in the gerbil hippocampus.
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Interaction between adenosine A1 and A2 receptor-mediated responses in the rat hippocampus in vitro.
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Sublethal in vitro glucose-oxygen deprivation protects cultured hippocampal neurons against a subsequent severe insult.
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Protection against kainate-induced excitotoxicity by adenosine A2A receptor agonists and antagonists.
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Effect of adenosine and some of its structural analogues on the conductance of NMDA receptor channels in a subset of rat neostriatal neurones.
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