Pre-conditioning protection in the brain.
作者信息
Stone T W
机构信息
Institute of Biomedical and Life Sciences, West Medical Building, University of Glasgow, Glasgow G12 8QQ, U.K.
出版信息
Br J Pharmacol. 2003 Sep;140(2):229-30. doi: 10.1038/sj.bjp.0705441. Epub 2003 Aug 11.
Abstract
摘要
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Pre-conditioning protection in the brain.大脑中的预处理保护
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Br J Pharmacol. 2003 Sep;140(2):305-14. doi: 10.1038/sj.bjp.0705442. Epub 2003 Aug 11.
2
The selective A2A receptor antagonist SCH 58261 reduces striatal transmitter outflow, turning behavior and ischemic brain damage induced by permanent focal ischemia in the rat.选择性A2A受体拮抗剂SCH 58261可减少大鼠永久性局灶性缺血诱导的纹状体递质释放、旋转行为和缺血性脑损伤。
Brain Res. 2003 Jan 10;959(2):243-50. doi: 10.1016/s0006-8993(02)03753-8.
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Suppression of presynaptic responses to adenosine by activation of NMDA receptors.通过NMDA受体激活抑制对腺苷的突触前反应。
Eur J Pharmacol. 2001 Sep 7;427(1):13-25. doi: 10.1016/s0014-2999(01)01171-2.
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Long-term potentiation protects rat hippocampal slices from the effects of acute hypoxia.长期增强作用可保护大鼠海马切片免受急性缺氧的影响。
Brain Res. 2001 Jul 13;907(1-2):144-50. doi: 10.1016/s0006-8993(01)02594-x.
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NMDA-induced changes in a cortical network in vivo are prevented by AMPA.AMPA可防止NMDA诱导的体内皮质网络变化。
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NMDA receptor antagonism, but not AMPA receptor antagonism attenuates induced ischaemic tolerance in the gerbil hippocampus.N-甲基-D-天冬氨酸（NMDA）受体拮抗作用而非α-氨基-3-羟基-5-甲基-4-异恶唑丙酸（AMPA）受体拮抗作用可减轻沙鼠海马体中的诱导性缺血耐受。
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Interaction between adenosine A1 and A2 receptor-mediated responses in the rat hippocampus in vitro.大鼠海马体中腺苷A1和A2受体介导反应的体外相互作用
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