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Bacterial invasion.

作者信息

DRUETT H A

出版信息

Nature. 1952 Aug 16;170(4320):288. doi: 10.1038/170288a0.

DOI:10.1038/170288a0
PMID:12993144
Abstract
摘要

相似文献

1
Bacterial invasion.细菌入侵
Nature. 1952 Aug 16;170(4320):288. doi: 10.1038/170288a0.
2
Host cellular unfolded protein response signaling regulates Campylobacter jejuni invasion.宿主细胞未折叠蛋白反应信号调节空肠弯曲菌的入侵。
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5
Apoptosis of human intestinal epithelial cells after bacterial invasion.细菌入侵后人类肠道上皮细胞的凋亡
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InvS Coordinates Expression of PrgH and FimZ and Is Required for Invasion of Epithelial Cells by Salmonella enterica serovar Typhimurium.InvS 协调 PrgH 和 FimZ 的表达，是鼠伤寒沙门氏菌侵袭上皮细胞所必需的。
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Dose-dependent interaction of parasites with tiers of host defense predicts "wormholes" that prolong infection at intermediate inoculum sizes.寄生虫与宿主防御层级之间的剂量依赖性相互作用预示着“虫洞”，即在中等接种量时会延长感染时间。
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5
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6
Stochastic bacterial population dynamics restrict the establishment of antibiotic resistance from single cells.随机细菌种群动态限制了单个细胞对抗生素耐药性的建立。
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Making pathogens sociable: the [corrected] emergence of high relatedness through limited host invasibility.让病原体变得“社交化”：通过有限的宿主可入侵性实现高亲缘关系的[修正后]出现。
ISME J. 2015 Oct;9(10):2315-23. doi: 10.1038/ismej.2015.111. Epub 2015 Jun 30.
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Bacterial Cooperation Causes Systematic Errors in Pathogen Risk Assessment due to the Failure of the Independent Action Hypothesis.由于独立作用假说不成立，细菌合作导致病原体风险评估中出现系统性错误。
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10
Unveiling time in dose-response models to infer host susceptibility to pathogens.剂量反应模型中的揭示时间，用于推断宿主对病原体的易感性。
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