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补体受体、黏附与吞噬作用。

Complement receptors, adhesion, and phagocytosis.

作者信息

Brown E J

机构信息

Division of Infectious Diseases, Washington University School of Medicine, St. Louis, MO 63110.

出版信息

Infect Agents Dis. 1992 Apr;1(2):63-70.

PMID:1365532
Abstract

Phagocytosis is an essential component of host defense against invading pathogens. Although many cell types can ingest particulate material, "professional phagocytes" perform this function much more efficiently than other cells because they express unique membrane proteins and signal transduction mechanisms. This review addresses the current state of knowledge about the process of phagocytosis and its regulation.

摘要

吞噬作用是宿主抵御入侵病原体的重要组成部分。尽管许多细胞类型都能摄取颗粒物质，但“专职吞噬细胞”执行此功能的效率比其他细胞高得多，因为它们表达独特的膜蛋白和信号转导机制。本文综述了吞噬作用及其调控过程的当前知识状态。

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Complement receptors, adhesion, and phagocytosis.补体受体、黏附与吞噬作用。
Infect Agents Dis. 1992 Apr;1(2):63-70.
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Phagocytosis of Mycobacterium tuberculosis is mediated by human monocyte complement receptors and complement component C3.结核分枝杆菌的吞噬作用由人单核细胞补体受体和补体成分C3介导。
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High concentrations of bacterial lipopolysaccharide, but not microbial infection-induced inflammation, activate macrophage C3 receptors for phagocytosis.高浓度的细菌脂多糖可激活巨噬细胞的C3受体以进行吞噬作用，但微生物感染诱导的炎症则不能。
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Functional aspects of the C1q receptors.C1q受体的功能方面。
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10
C1q interactions with cell surface receptors.C1q与细胞表面受体的相互作用。
Behring Inst Mitt. 1989 Jul(84):220-9.

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