HARTLEY J W, ROWE W P, CHANOCK R M, ANDREWS B E
J Exp Med. 1959 Jul 1;110(1):81-91. doi: 10.1084/jem.110.1.81.
Treatment of guinea pig erythrocytes with types A and B influenza viruses rendered them inagglutinable by polyoma virus; also, the inhibitory effect of ovomucin on polyoma virus hemagglutination was destroyed by pretreatment of the ovomucin with various myxoviruses. These results indicate that polyoma virus and myxovirus erythrocyte receptor sites are identical. However, no destruction by polyoma virus of its own or of myxovirus receptors or inhibitors was detected. No serologic relationship was detected between polyoma virus and members of the myxovirus group; differences in size and stability further indicate their distinctness. No evidence was found of biologic or serologic relationship of polyoma virus with encephalomyocarditis virus or mouse encephalomyelitis virus.
用甲型和乙型流感病毒处理豚鼠红细胞，使其不能被多瘤病毒凝集；此外，通过用各种黏液病毒对卵黏蛋白进行预处理，卵黏蛋白对多瘤病毒血凝反应的抑制作用被破坏。这些结果表明，多瘤病毒和黏液病毒的红细胞受体位点是相同的。然而，未检测到多瘤病毒对其自身或黏液病毒受体或抑制剂的破坏作用。未检测到多瘤病毒与黏液病毒组各成员之间的血清学关系；大小和稳定性的差异进一步表明它们是不同的。未发现多瘤病毒与脑心肌炎病毒或小鼠脑脊髓炎病毒存在生物学或血清学关系的证据。
