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The delayed origin of mutants induced by exposure of extracellular phage T4 to ethyl methane sulfonate.通过将细胞外噬菌体T4暴露于甲磺酸乙酯而诱导的突变体的延迟起源。
Proc Natl Acad Sci U S A. 1961 Jan 15;47(1):64-72. doi: 10.1073/pnas.47.1.64.
2
The nature of the "deletion" mutants in the rII region of phage T4.噬菌体T4 rII区域中“缺失”突变体的性质。
J Mol Biol. 1961 Oct;3:684-92. doi: 10.1016/s0022-2836(61)80031-4.
3
Mutations affecting the lysozyme of phage T4.影响噬菌体T4溶菌酶的突变
Cold Spring Harb Symp Quant Biol. 1961;26:25-30. doi: 10.1101/sqb.1961.026.01.007.
4
Early enzyme synthesis and its control in E. coli infected with some amber mutants of bacteriophage T4.用噬菌体T4的一些琥珀突变体感染大肠杆菌后早期酶的合成及其调控
Proc Natl Acad Sci U S A. 1962 Feb;48(2):293-302. doi: 10.1073/pnas.48.2.293.
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The influence of radiomimetic substances on deoxyribonucleic acid synthesis and function studies in Escherichia coli/phage systems. I. The nature of the inactivation of T2 phage in vitro by certain alkylating agents.拟放射性物质对大肠杆菌/噬菌体系统中脱氧核糖核酸合成及功能的影响。I. 某些烷基化剂在体外对T2噬菌体的灭活性质。
Proc R Soc Lond B Biol Sci. 1959 Apr 21;150(940):423-45. doi: 10.1098/rspb.1959.0032.
6
Further studies on induced reverse mutations of phage T4 rII mutants.
Biochem Biophys Res Commun. 1962 Feb 20;7:18-22. doi: 10.1016/0006-291x(62)90136-5.
7
[The cotransduction of pET system plasmids by mutants of T4 and RB43 bacteriophages].[T4和RB43噬菌体突变体对pET系统质粒的共转导]
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8
Induction of reverse mutations and cross reactivation of nitrous acid-treated phage T4.亚硝酸处理的噬菌体T4的回复突变诱导及交叉复活
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9
Engineering nucleic acid-binding proteins by phage display.通过噬菌体展示技术工程化核酸结合蛋白。
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10
Formation of non-phage-antigenic protein in E. coli infected with T2 phage.被T2噬菌体感染的大肠杆菌中非噬菌体抗原蛋白的形成。
Biochim Biophys Acta. 1957 Sep;25(3):665-6. doi: 10.1016/0006-3002(57)90552-8.

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Engineering Phages to Fight Multidrug-Resistant Bacteria.改造噬菌体以对抗多重耐药细菌。
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Allele-specific suppression in Caenorhabditis elegans reveals details of EMS mutagenesis and a possible moonlighting interaction between the vesicular acetylcholine transporter and ERD2 receptors.在秀丽隐杆线虫中，等位基因特异性抑制揭示了 EMS 诱变的细节，以及囊泡乙酰胆碱转运体和 ERD2 受体之间可能的兼职相互作用。
Genetics. 2021 Aug 9;218(4). doi: 10.1093/genetics/iyab065.
3
Mutagenesis at a complex locus in Drosophila with the monofunctional alkylating agent, ethyl methanesulfonate.利用单功能烷基化剂甲磺酸乙酯在果蝇的一个复杂基因座上进行诱变。
Genetics. 1967 Dec;57(4):783-93. doi: 10.1093/genetics/57.4.783.
4
Ultraviolet mutagenesis in bacteriophage T-4. I. Irradiation of extracellular phage particles.噬菌体T-4中的紫外线诱变。I. 细胞外噬菌体颗粒的辐照。
J Bacteriol. 1966 May;91(5):1775-80. doi: 10.1128/jb.91.5.1775-1780.1966.
5
Genotypic reversion by methylene blue: the orientation of guanine-hydroxymethylcytosine at mutated sites in rII mutants of phage T4.
Mol Gen Genet. 1968;101(1):51-8. doi: 10.1007/BF00434811.
6
UV-induced mutation in bacteriophage T4.紫外线诱导的噬菌体T4突变。
J Virol. 1978 May;26(2):265-71. doi: 10.1128/JVI.26.2.265-271.1978.

本文引用的文献

1
Phenotypic Properties of Heterozygotes in the Bacteriophage T4.噬菌体T4杂合子的表型特性
Genetics. 1958 Mar;43(2):235-48. doi: 10.1093/genetics/43.2.235.
2
MUTATIONAL HETEROZYGOTES IN BACTERIOPHAGES.噬菌体中的突变杂合子
Proc Natl Acad Sci U S A. 1959 Oct;45(10):1507-15. doi: 10.1073/pnas.45.10.1507.
3
The frequency distribution of spontaneous bacteriophage mutants as evidence for the exponential rate of phage reproduction.自发噬菌体突变体的频率分布作为噬菌体繁殖指数率的证据。
Cold Spring Harb Symp Quant Biol. 1951;16:463-70. doi: 10.1101/sqb.1951.016.01.033.
4
Induced reversion of T4rII mutants by ultraviolet irradiation of extracellular phage.通过对细胞外噬菌体进行紫外线照射诱导T4rII突变体的回复突变
Virology. 1959 Oct;9:215-27. doi: 10.1016/0042-6822(59)90116-3.
5
Increased rate of plaque-type and host-range mutation following treatment of bacteriophage in vitro with ethyl methane sulphonate.用甲磺酸乙酯对噬菌体进行体外处理后，噬菌斑类型和宿主范围突变率增加。
Nature. 1958 Apr 26;181(4617):1212-3. doi: 10.1038/1811212a0.
6
The structure of DNA.DNA的结构。
Cold Spring Harb Symp Quant Biol. 1953;18:123-31. doi: 10.1101/sqb.1953.018.01.020.

The delayed origin of mutants induced by exposure of extracellular phage T4 to ethyl methane sulfonate.

作者信息

GREEN D M, KRIEG D R

出版信息

Proc Natl Acad Sci U S A. 1961 Jan 15;47(1):64-72. doi: 10.1073/pnas.47.1.64.

DOI:10.1073/pnas.47.1.64
PMID:13708170
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC285235/
Abstract
摘要