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使用布雷菲德菌素A定位水泡性口炎病毒G蛋白在干扰素处理细胞内的转运阻滞。

Use of Brefeldin A to localize block in intracellular transport of vesicular stomatitis virus G protein on interferon-treated cells.

作者信息

Polakova K, Russ G

机构信息

Cancer Research Institute, Slovak Academy of Sciences, Bratislava, Czechoslovakia.

出版信息

Arch Virol. 1992;124(1-2):171-9. doi: 10.1007/BF01314635.

DOI:10.1007/BF01314635
PMID:1373940
Abstract

Brefeldin A (BFA) induced a rapid redistribution of vesicular stomatitis virus G protein (VSV-G) to the endoplasmatic reticulum (ER) in interferon (IFN)-pretreated cells. This result is consistent with accumulation of VSV-G in the trans-Golgi (GC) complex in cells pretreated with IFN and implies that IFN does not interfere with the ability of BFA to induce redistribution of proteins from GC to ER.

摘要

布雷菲德菌素A（BFA）可诱导水泡性口炎病毒G蛋白（VSV-G）在经干扰素（IFN）预处理的细胞中迅速重新分布至内质网（ER）。这一结果与VSV-G在经IFN预处理的细胞中转高尔基体（GC）复合体中的积累情况一致，意味着IFN并不干扰BFA诱导蛋白质从GC重新分布至ER的能力。

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Use of Brefeldin A to localize block in intracellular transport of vesicular stomatitis virus G protein on interferon-treated cells.使用布雷菲德菌素A定位水泡性口炎病毒G蛋白在干扰素处理细胞内的转运阻滞。
Arch Virol. 1992;124(1-2):171-9. doi: 10.1007/BF01314635.
2
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J Virol. 1993 Dec;67(12):7533-8. doi: 10.1128/JVI.67.12.7533-7538.1993.
3
Interferon alters intracellular transport of vesicular stomatitis virus glycoprotein.干扰素改变水疱性口炎病毒糖蛋白的细胞内运输。
J Biol Regul Homeost Agents. 1988 Apr-Jun;2(2):53-62.
4
Post-Golgi membrane traffic: brefeldin A inhibits export from distal Golgi compartments to the cell surface but not recycling.高尔基体后膜转运：布雷菲德菌素A抑制从高尔基体远端区室向细胞表面的转运，但不抑制再循环。
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Dissociation of coatomer from membranes is required for brefeldin A-induced transfer of Golgi enzymes to the endoplasmic reticulum.衣被蛋白从膜上解离是布雷菲德菌素A诱导高尔基体酶向内质网转移所必需的。
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Brefeldin A redistributes resident and itinerant Golgi proteins to the endoplasmic reticulum.布雷菲德菌素A将驻留型和循环型高尔基体蛋白重新分布到内质网。
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本文引用的文献

1
Effect of interferon treatment on vesicular stomatitis virus (VSV): release of unusual particles with low infectivity.干扰素治疗对水疱性口炎病毒（VSV）的影响：释放出感染性低的异常颗粒。
Virology. 1980 Mar;101(2):399-407. doi: 10.1016/0042-6822(80)90453-5.
2
Interferon induces the production of membrane protein-deficient and infectivity-defective vesicular stomatitis virions through interference in the virion assembly process.干扰素通过干扰病毒粒子的装配过程，诱导产生膜蛋白缺陷且感染性有缺陷的水疱性口炎病毒粒子。
J Gen Virol. 1983 Mar;64 Pt 3:707-12. doi: 10.1099/0022-1317-64-3-707.
3
Interferon-treated cells release vesicular stomatitis virus particles lacking glycoprotein spikes: correlation with biochemical data.
经干扰素处理的细胞释放缺乏糖蛋白刺突的水疱性口炎病毒颗粒：与生化数据的相关性。
Proc Natl Acad Sci U S A. 1980 Apr;77(4):2284-7. doi: 10.1073/pnas.77.4.2284.
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Effect of cloned human interferons on protein synthesis and morphogenesis of herpes simplex virus.克隆的人干扰素对单纯疱疹病毒蛋白质合成及形态发生的影响
J Virol. 1985 Nov;56(2):419-25. doi: 10.1128/JVI.56.2.419-425.1985.
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Targeting and processing of glycophorins in murine erythroleukemia cells: use of brefeldin A as a perturbant of intracellular traffic.小鼠红白血病细胞中血型糖蛋白的靶向与加工：使用布雷菲德菌素A作为细胞内运输的干扰剂
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6
Brefeldin A redistributes resident and itinerant Golgi proteins to the endoplasmic reticulum.布雷菲德菌素A将驻留型和循环型高尔基体蛋白重新分布到内质网。
J Cell Biol. 1989 Jul;109(1):61-72. doi: 10.1083/jcb.109.1.61.
7
Rapid redistribution of Golgi proteins into the ER in cells treated with brefeldin A: evidence for membrane cycling from Golgi to ER.用布雷菲德菌素A处理的细胞中高尔基体蛋白快速重新分布到内质网：高尔基体到内质网的膜循环证据
Cell. 1989 Mar 10;56(5):801-13. doi: 10.1016/0092-8674(89)90685-5.
8
Defective transport of hemagglutinin-neuraminidase glycoprotein of bovine parainfluenza-3 virus in interferon treated cell.牛副流感3型病毒血凝素神经氨酸酶糖蛋白在干扰素处理的细胞中的转运缺陷
Arch Virol. 1989;109(1-2):125-33. doi: 10.1007/BF01310524.
9
Interferon alters intracellular transport of vesicular stomatitis virus glycoprotein.干扰素改变水疱性口炎病毒糖蛋白的细胞内运输。
J Biol Regul Homeost Agents. 1988 Apr-Jun;2(2):53-62.
10
Microtubule-dependent retrograde transport of proteins into the ER in the presence of brefeldin A suggests an ER recycling pathway.在布雷菲德菌素A存在的情况下，蛋白质通过微管依赖的逆行转运进入内质网，这表明存在内质网再循环途径。
Cell. 1990 Mar 9;60(5):821-36. doi: 10.1016/0092-8674(90)90096-w.