CARROLL R
J Clin Pathol. 1963 Mar;16(2):133-6. doi: 10.1136/jcp.16.2.133.
The rarity of hepatic infarction in the human being is commonly explained as due to the double blood supply via the hepatic artery and the portal vein. This explanation cannot be accepted, because, if the arterial blood supply alone is arrested the portal blood supply does not protect the liver from infarction. The factor of importance is that the arterial blood supply comes by three different routes and it is difficult to obstruct these collateral supplies simultaneously. Infarction of the liver can be caused by thrombosis or ligation of the main hepatic artery between the origin of the right gastric artery and the hilum of the liver, but the more common causes are infected emboli or polyarteritis nodosa; these presumably interfere not only with the main arterial supply but also with some of the collateral arterial supply.
人类肝梗死罕见通常被解释为是由于肝动脉和门静脉的双重血液供应。这种解释是不能被接受的，因为，如果仅阻断动脉血液供应，门静脉血液供应并不能保护肝脏免于梗死。重要的因素是动脉血液供应通过三条不同途径而来，同时阻断这些侧支供应是困难的。肝梗死可由右胃动脉起始处与肝门之间的肝总动脉血栓形成或结扎引起，但更常见的原因是感染性栓子或结节性多动脉炎；这些情况大概不仅干扰了主要动脉供应，而且还干扰了一些侧支动脉供应。
