Duckwiler G R, Dion J E, Viñuela F, Reichman A
Department of Radiological Services, UCLA School of Medicine 90024.
AJNR Am J Neuroradiol. 1992 Nov-Dec;13(6):1571-9.
To describe the nature and features of delayed venous thrombosis following embolization of arteriovenous fistulae.
Retrospective review of the available clinical history, details of embolization procedures, and results of follow-up angiography were carried out on all embolization procedures performed on high-flow vascular malformations of the brain done at our institution since 1987.
Four patients were identified who had delayed (greater than 1 week) venous thrombosis/occlusion after embolization of the malformation associated with neurologic complications. Two patients had arteriovenous fistula and two had vein-of-Galen malformations. These patients had no untoward embolization of the venous outlet as a cause of the venous occlusion.
It is postulated that thrombosis in the arteriovenous fistula group was induced by conversion (due to embolization) of a patulous high flow venous outlet into a slow flow system; in the vein-of-Galen group, the occlusion was thought to be due to high-flow venopathy.
描述动静脉瘘栓塞术后延迟性静脉血栓形成的性质和特征。
对1987年以来在本机构进行的所有脑高流量血管畸形栓塞手术的现有临床病史、栓塞手术细节及随访血管造影结果进行回顾性分析。
4例患者在畸形栓塞后出现延迟性（超过1周）静脉血栓形成/闭塞，并伴有神经并发症。其中2例为动静脉瘘，2例为大脑大静脉畸形。这些患者的静脉闭塞并非由静脉出口不良栓塞所致。
推测动静脉瘘组的血栓形成是由于扩张的高流量静脉出口（因栓塞）转变为低流量系统所致；在大脑大静脉组，闭塞被认为是由于高流量静脉病变所致。
