Lin Peter H, Bush Ruth L, Katzman John B, Zemel Gerald, Puente Orlando A, Katzen Barry T, Lumsden Alan B
Division of Vascular Surgery and Endovascular Therapy, DeBakey Department of Surgery, Baylor College of Medicine, Houston VAMC (112), 2002 Holcomb Boulevard, Houston, TX 77030, USA.
J Vasc Surg. 2003 Oct;38(4):840-2. doi: 10.1016/s0741-5214(03)00468-3.
Abdominal aortic aneurysm (AAA) enlarges after successful endovascular repair, because of persistent blood flow within the aneurysm sac, or endoleak. In the absence of detectable endoleak, AAA may still expand, in part because of persistent pressurization within the excluded aneurysm, or endotension. We report three patients who underwent successful endovascular AAA repair in whom postoperative surveillance showed aneurysm regression, yet delayed AAA enlargement without demonstrable endoleak developed in all three patients. Endotension was confirmed in all three patients at elective open conversion. Our study underscores the significance of endotension as a mechanism of delayed aneurysm enlargement after successful endovascular AAA repair.
腹主动脉瘤(AAA)在成功进行血管腔内修复后仍会扩大,原因是瘤腔内持续存在血流,即内漏。在未检测到内漏的情况下,AAA仍可能扩大,部分原因是被排除的动脉瘤内持续存在压力,即内张力。我们报告了3例成功进行血管腔内AAA修复的患者,术后监测显示动脉瘤缩小,但所有3例患者均出现了无明显内漏的延迟性AAA扩大。在择期开放转换时,所有3例患者均证实存在内张力。我们的研究强调了内张力作为成功进行血管腔内AAA修复后延迟性动脉瘤扩大机制的重要性。